Pages

Friday, October 7, 2011

Body Condition Scoring for Cats

Bjornvad CR, Nielsen DH, Armstrong PJ et al: Evaluation of a nine-point body condition scoring system in physically inactive pet cats, Am J Vet Res 72:433, 2011.

About 35% to 40% of the adult pet cat population is overweight or obese. Obesity in cats predisposes them to diseases such as lower urinary tract disease, lameness, hepatic lipidosis, non-allergic skin disease, and diabetes mellitus. Body weight (BW) is an objective measurement and is useful for follow-up in weight loss programs, but it does not provide any indication of body composition or lean-to-fat body mass. The most widely used system for body composition is the 9-point semi-quantitative system for body condition score (BCS) that is based on visual and palpatory findings (BCS: 1 = emaciated, 5 = ideal, and 9 = grossly obese). The purpose of this study was to evaluate whether a 9-point BCS system and the % body fat (BF) assigned to each BCS based on data from colony cats is valid for privately owned, indoor-confined, adult neutered DSH cats. The study determined that the 9-point BCS system reflected actual body composition well in pet DSH cats. Nevertheless, the percentage of BF associated with each BCS was higher in this study than was reported for colony cats. This was particularly noted for male cats. For relevancy to veterinary practice, it was recommended that to detect subtle weight change in individual cats, the BCS system should be combined with regular BW measurements at all veterinary visits. Study findings could indicate a need for redefining the ideal BCS for inactive neutered cats to include a BCS of 4. [VT]

Related articles:
Baez JL, Michel KE, Sorenmo K et al: A prospective investigation of the prevalence and prognostic significance of weight loss and changes in body condition in feline cancer patients, J Feline Med Surg 9:411, 2007.

More on cat health: Winn Feline Foundation Library
Join us on Facebook
Follow us on Twitter
Bjornvad CR, Nielsen DH, Armstrong PJ et al: Evaluation of a nine-point body condition scoring system in physically inactive pet cats, Am J Vet Res 72:433, 2011.

About 35% to 40% of the adult pet cat population is overweight or obese. Obesity in cats predisposes them to diseases such as lower urinary tract disease, lameness, hepatic lipidosis, non-allergic skin disease, and diabetes mellitus. Body weight (BW) is an objective measurement and is useful for follow-up in weight loss programs, but it does not provide any indication of body composition or lean-to-fat body mass. The most widely used system for body composition is the 9-point semi-quantitative system for body condition score (BCS) that is based on visual and palpatory findings (BCS: 1 = emaciated, 5 = ideal, and 9 = grossly obese). The purpose of this study was to evaluate whether a 9-point BCS system and the % body fat (BF) assigned to each BCS based on data from colony cats is valid for privately owned, indoor-confined, adult neutered DSH cats. The study determined that the 9-point BCS system reflected actual body composition well in pet DSH cats. Nevertheless, the percentage of BF associated with each BCS was higher in this study than was reported for colony cats. This was particularly noted for male cats. For relevancy to veterinary practice, it was recommended that to detect subtle weight change in individual cats, the BCS system should be combined with regular BW measurements at all veterinary visits. Study findings could indicate a need for redefining the ideal BCS for inactive neutered cats to include a BCS of 4. [VT]

Related articles:
Baez JL, Michel KE, Sorenmo K et al: A prospective investigation of the prevalence and prognostic significance of weight loss and changes in body condition in feline cancer patients, J Feline Med Surg 9:411, 2007.

More on cat health: Winn Feline Foundation Library
Join us on Facebook
Follow us on Twitter
Read More


Tuesday, October 4, 2011

Feline Parvovirus

Battilani M, Balboni A, Ustulin M et al: Genetic complexity and multiple infections with more Parvovirus species in naturally infected cats, Veterinary research 42:43, 2011.

The parvoviruses affecting dogs and cats are very closely related and cause similar disease. Several strains of canine parvovirus (CPV) can infect and cause disease in cats, including CPV-2a, -2b, and -2c. This opens up the possibility that a cat could be infected with feline parvovirus (FPV) as well as CPV. These researchers in Italy analyzed the parvoviruses infecting 24 cats. While 22 of the cats had FPV, there was evidence of CPV infection in two of the cats. One cat had CPV-2c, while the other had both FPV and CPV-2a – coinfection with two different viruses. In addition, it was noted that the CPV strains infecting both cats were relatively genetically diverse; that is, these viruses appeared to be re-adapting to the feline host. The researchers speculate that infected cats could potentially be a source of new parvovirus variants. [MK]

Related articles:

Kruse BD, Unterer S, Horlacher K et al: Prognostic factors in cats with feline panleukopenia, J Vet Intern Med 24:1271, 2010.

More on cat health: Winn Feline Foundation Library
Join us on Facebook
Follow us on Twitter
Battilani M, Balboni A, Ustulin M et al: Genetic complexity and multiple infections with more Parvovirus species in naturally infected cats, Veterinary research 42:43, 2011.

The parvoviruses affecting dogs and cats are very closely related and cause similar disease. Several strains of canine parvovirus (CPV) can infect and cause disease in cats, including CPV-2a, -2b, and -2c. This opens up the possibility that a cat could be infected with feline parvovirus (FPV) as well as CPV. These researchers in Italy analyzed the parvoviruses infecting 24 cats. While 22 of the cats had FPV, there was evidence of CPV infection in two of the cats. One cat had CPV-2c, while the other had both FPV and CPV-2a – coinfection with two different viruses. In addition, it was noted that the CPV strains infecting both cats were relatively genetically diverse; that is, these viruses appeared to be re-adapting to the feline host. The researchers speculate that infected cats could potentially be a source of new parvovirus variants. [MK]

Related articles:

Kruse BD, Unterer S, Horlacher K et al: Prognostic factors in cats with feline panleukopenia, J Vet Intern Med 24:1271, 2010.

More on cat health: Winn Feline Foundation Library
Join us on Facebook
Follow us on Twitter
Read More


Monday, October 3, 2011

Scoliosis: Straight Talk About Twisted Spines

By Brett Diaz, DC


Although most chiropractors treat patients who have scoliosis, few truly understand the forces that lock up these abnormal curves.


This can make treating a scoliosis curvature feel like trying to treat a spine cast in bronze. The good news is it is not that rigid. The key to reshaping the spine toward a more normal, balanced position is to first understand the pathway it took to get so twisted.
Although a two-dimensional radiograph will quite readily reveal the lateral curves of scoliosis, because it is not a three-dimensional representation of the body it does nothing to reveal the all-important rotational component most often found in scoliosis. One of the greatest challenges in treating scoliosis is for the practitioner to truly appreciate the full depth of the problem. This difficulty lies greatly in the limitations of the primary diagnostic tool used for scoliosis, the plain-film X-ray. Although X-rays of a scoliosis are quite dramatic, they are still just a two-dimensional representation of an even more complex three-dimensional problem. Thus, simply viewing X-rays of a scoliosis often draws the attention of the doctor to only the two obvious dimensions of the spine, the vertical height and the lateral deviation. This focus results in significant visual limitations for doctor because it fails to capture the third dimension of the scoliosis.

Although scoliosis is easy to see on plain-film X-rays, the unfortunate reality is that this two-dimensional view does not show one of the key components that must be addressed in order to help improve scoliosis. Understanding this "unseen element" will provide useful insights into the best techniques that can help you enhance your treatment of scoliosis and help you obtain better clinical outcomes. By understanding and providing the proper kind of corrections, you can assist your scoliosis patients in stopping the progression of their curves and in many cases actually help them reduce their curves.
The difference in the level of conceptual understanding of scoliosis of the spine can best be illustrated by considering the differences between a two- and three-dimensional chess board. Although the classical game of chess is played in only two dimensions, it is still an intellectually challenging game that requires a great deal of global thinking to strategize how the various combinations of pieces interact on the board. Now imagine just how much more thinking is required in a game of chess on a three-dimensional board (for example, as portrayed on many episodes of "Star Trek"). Instead of just thinking about moving pieces in two planes, consider attacks and counterattacks in a third dimension. This adds a whole other level of complexity to your play. The same increased level of conceptual thinking is required to effectively treat a three-dimensional scoliosis.
Appreciating the Unseen Core Element of Scoliosis
When they are viewed on X-ray, scoliosis curves appear "S" shaped and span both the thoracic and lumbar spine; however, the reality is that there is far more to this curve than just meets the eye. Because X-rays are only two-dimensional representations of a three-dimensional body, what looks like a flattened-out "S" curve on an X-ray is in reality more like the helix of a spring in the body. Understanding this distinction is critical when treating the spine to stop the progression of or correct a scoliosis curvature.
This helix-shaped curvature is caused by a unilateral rotation dysfunction of the deep postural muscles surrounding the spine. There is a neurological basis to this imbalance and it is at the heart of the scoliosis. Correction and stabilization of these muscular imbalances must be addressed in order to stop progression of the curve.
Unfortunately, this three-dimensional rotational twist is often overlooked during the treatment process, which can cause dire consequences. When a chiropractor does not take this rotational component into consideration and applies nonspecific bilateral treatment to the spine, they can actually cause an increase in the curvature by contributing to the unilateral rotation dysfunction; therefore, this type of generalized adjusting is contraindicated for scoliosis.
While a rotatory force applied in the opposite direction of the torque will help correct a unilateral rotation dysfunction, doing adjustments in the direction of the scoliosis spiral will actually serve to reinforce the abnormal movement patterns and end up worsening the condition. Additionally, adjustments that are aimed solely on straightening the concave side of the curve in an attempt to reduce a two-dimensional Cobb angle on X-ray can also be detrimental if the aforementioned rotation component is not taken into consideration.
Take a Global Approach to Help Your Scoliosis Patients
Another analogy to help illustrate the inadequacy of this two-dimensional approach is to think of trying to straighten out a metal spring. Much like a spring, the spine in scoliosis is in a coil formation that not only curves side to side, but also twists forward and back. If all you are trying to do is bend it from side to side, you are never going to be able to effectively straighten it unless you also address the spiral bend in the coil first.
Because the scoliosis curvature is simply part of a global neurological pattern that affects the entire body, attempts to just "fix" local-level curvatures are doomed to achieve only short-lived results. This larger neurological element must be properly addressed if you expect to halt the progression of a scoliosis curve, especially in the rapid growth phase just before puberty.
A final analogy that's important to consider when managing scoliosis patients: Think of the scoliosis spine as a wet washcloth that is being wrung out. As it twists, it shortens and buckles on itself. The spine acts the same way. Each vertebral segment tends to lock out in 4 degrees of rotation. So, to straighten out the lateral curve, you first have to simultaneously "unwring" it by releasing the shortened and restricted rotational muscles of the spine while simultaneously unloading the spine axially. Once you have corrected this rotational dysfunction and strengthened the appropriate antagonist muscle, the lateral deformity caused by the collapsing of the spine will improve and you will experience greater success and more lasting results with your scoliosis patients.
Scoliosis on Two-Dimensional X-Ray – A Limited View


Although a two-dimensional radiograph will quite readily reveal the lateral curves of scoliosis, because it is not a three-dimensional representation of the body it does nothing to reveal the all-important rotational component most often found in scoliosis. One of the greatest challenges in treating scoliosis is for the practitioner to truly appreciate the full depth of the problem. This difficulty lies greatly in the limitations of the primary diagnostic tool used for scoliosis, the plain-film X-ray. Although X-rays of a scoliosis are quite dramatic, they are still just a two-dimensional representation of an even more complex three-dimensional problem. Thus, simply viewing X-rays of a scoliosis often draws the attention of the doctor to only the two obvious dimensions of the spine, the vertical height and the lateral deviation. This focus results in significant visual limitations for doctor because it fails to capture the third dimension of the scoliosis.
The difference in the level of conceptual understanding of scoliosis of the spine can best be illustrated by considering the differences between a two- and three-dimensional chess board. Although the classical game of chess is played in only two dimensions, it is still an intellectually challenging game that requires a great deal of global thinking to strategize how the various combinations of pieces interact on the board. Now imagine just how much more thinking is required in a game of chess on a three-dimensional board (for example, as portrayed on many episodes of "Star Trek"). Instead of just thinking about moving pieces in two planes, consider attacks and counterattacks in a third dimension. This adds a whole other level of complexity to your play. The same increased level of conceptual thinking is required to effectively treat a three-dimensional scoliosis.
Appreciating the Unseen Core Element of Scoliosis
When they are viewed on X-ray, scoliosis curves appear "S" shaped and span both the thoracic and lumbar spine; however, the reality is that there is far more to this curve than just meets the eye. Because X-rays are only two-dimensional representations of a three-dimensional body, what looks like a flattened-out "S" curve on an X-ray is in reality more like the helix of a spring in the body. Understanding this distinction is critical when treating the spine to stop the progression of or correct a scoliosis curvature.
This helix-shaped curvature is caused by a unilateral rotation dysfunction of the deep postural muscles surrounding the spine. There is a neurological basis to this imbalance and it is at the heart of the scoliosis. Correction and stabilization of these muscular imbalances must be addressed in order to stop progression of the curve.
Unfortunately, this three-dimensional rotational twist is often overlooked during the treatment process, which can cause dire consequences. When a chiropractor does not take this rotational component into consideration and applies nonspecific bilateral treatment to the spine, they can actually cause an increase in the curvature by contributing to the unilateral rotation dysfunction; therefore, this type of generalized adjusting is contraindicated for scoliosis.
While a rotatory force applied in the opposite direction of the torque will help correct a unilateral rotation dysfunction, doing adjustments in the direction of the scoliosis spiral will actually serve to reinforce the abnormal movement patterns and end up worsening the condition. Additionally, adjustments that are aimed solely on straightening the concave side of the curve in an attempt to reduce a two-dimensional Cobb angle on X-ray can also be detrimental if the aforementioned rotation component is not taken into consideration.
Take a Global Approach to Help Your Scoliosis Patients
Another analogy to help illustrate the inadequacy of this two-dimensional approach is to think of trying to straighten out a metal spring. Much like a spring, the spine in scoliosis is in a coil formation that not only curves side to side, but also twists forward and back. If all you are trying to do is bend it from side to side, you are never going to be able to effectively straighten it unless you also address the spiral bend in the coil first.
Because the scoliosis curvature is simply part of a global neurological pattern that affects the entire body, attempts to just "fix" local-level curvatures are doomed to achieve only short-lived results. This larger neurological element must be properly addressed if you expect to halt the progression of a scoliosis curve, especially in the rapid growth phase just before puberty.
A final analogy that's important to consider when managing scoliosis patients: Think of the scoliosis spine as a wet washcloth that is being wrung out. As it twists, it shortens and buckles on itself. The spine acts the same way. Each vertebral segment tends to lock out in 4 degrees of rotation. So, to straighten out the lateral curve, you first have to simultaneously "unwring" it by releasing the shortened and restricted rotational muscles of the spine while simultaneously unloading the spine axially. Once you have corrected this rotational dysfunction and strengthened the appropriate antagonist muscle, the lateral deformity caused by the collapsing of the spine will improve and you will experience greater success and more lasting results with your scoliosis patients.

Dr. Brett Diaz is a 1987 graduate of Los Angeles College of Chiropractic (now Southern California University of Health Sciences). He is the director of clinical services for scoliosis treatment alternatives at O.C. Back & Body Doctors, in Irvine, Calif., where he specializes in nonsurgical treatment of scoliosis utilizing a variety of conservative modalities including chiropractic care, physical therapy, massage therapy, dynamic bracing, vibration therapy, scoliosis-specific exercises, active release techniques, Pilates, and de-rotation traction therapy. 

Dr. Brett Diaz is a 1987 graduate of Los Angeles College of Chiropractic (now Southern California University of Health Sciences). He is the director of clinical services for scoliosis treatment alternatives at O.C. Back & Body Doctors, in Irvine, Calif., where he specializes in nonsurgical treatment of scoliosis utilizing a variety of conservative modalities including chiropractic care, physical therapy, massage therapy, dynamic bracing, vibration therapy, scoliosis-specific exercises, active release techniques, Pilates, and de-rotation traction therapy. 
By Brett Diaz, DC


Although most chiropractors treat patients who have scoliosis, few truly understand the forces that lock up these abnormal curves.


This can make treating a scoliosis curvature feel like trying to treat a spine cast in bronze. The good news is it is not that rigid. The key to reshaping the spine toward a more normal, balanced position is to first understand the pathway it took to get so twisted.
Although a two-dimensional radiograph will quite readily reveal the lateral curves of scoliosis, because it is not a three-dimensional representation of the body it does nothing to reveal the all-important rotational component most often found in scoliosis. One of the greatest challenges in treating scoliosis is for the practitioner to truly appreciate the full depth of the problem. This difficulty lies greatly in the limitations of the primary diagnostic tool used for scoliosis, the plain-film X-ray. Although X-rays of a scoliosis are quite dramatic, they are still just a two-dimensional representation of an even more complex three-dimensional problem. Thus, simply viewing X-rays of a scoliosis often draws the attention of the doctor to only the two obvious dimensions of the spine, the vertical height and the lateral deviation. This focus results in significant visual limitations for doctor because it fails to capture the third dimension of the scoliosis.

Although scoliosis is easy to see on plain-film X-rays, the unfortunate reality is that this two-dimensional view does not show one of the key components that must be addressed in order to help improve scoliosis. Understanding this "unseen element" will provide useful insights into the best techniques that can help you enhance your treatment of scoliosis and help you obtain better clinical outcomes. By understanding and providing the proper kind of corrections, you can assist your scoliosis patients in stopping the progression of their curves and in many cases actually help them reduce their curves.
The difference in the level of conceptual understanding of scoliosis of the spine can best be illustrated by considering the differences between a two- and three-dimensional chess board. Although the classical game of chess is played in only two dimensions, it is still an intellectually challenging game that requires a great deal of global thinking to strategize how the various combinations of pieces interact on the board. Now imagine just how much more thinking is required in a game of chess on a three-dimensional board (for example, as portrayed on many episodes of "Star Trek"). Instead of just thinking about moving pieces in two planes, consider attacks and counterattacks in a third dimension. This adds a whole other level of complexity to your play. The same increased level of conceptual thinking is required to effectively treat a three-dimensional scoliosis.
Appreciating the Unseen Core Element of Scoliosis
When they are viewed on X-ray, scoliosis curves appear "S" shaped and span both the thoracic and lumbar spine; however, the reality is that there is far more to this curve than just meets the eye. Because X-rays are only two-dimensional representations of a three-dimensional body, what looks like a flattened-out "S" curve on an X-ray is in reality more like the helix of a spring in the body. Understanding this distinction is critical when treating the spine to stop the progression of or correct a scoliosis curvature.
This helix-shaped curvature is caused by a unilateral rotation dysfunction of the deep postural muscles surrounding the spine. There is a neurological basis to this imbalance and it is at the heart of the scoliosis. Correction and stabilization of these muscular imbalances must be addressed in order to stop progression of the curve.
Unfortunately, this three-dimensional rotational twist is often overlooked during the treatment process, which can cause dire consequences. When a chiropractor does not take this rotational component into consideration and applies nonspecific bilateral treatment to the spine, they can actually cause an increase in the curvature by contributing to the unilateral rotation dysfunction; therefore, this type of generalized adjusting is contraindicated for scoliosis.
While a rotatory force applied in the opposite direction of the torque will help correct a unilateral rotation dysfunction, doing adjustments in the direction of the scoliosis spiral will actually serve to reinforce the abnormal movement patterns and end up worsening the condition. Additionally, adjustments that are aimed solely on straightening the concave side of the curve in an attempt to reduce a two-dimensional Cobb angle on X-ray can also be detrimental if the aforementioned rotation component is not taken into consideration.
Take a Global Approach to Help Your Scoliosis Patients
Another analogy to help illustrate the inadequacy of this two-dimensional approach is to think of trying to straighten out a metal spring. Much like a spring, the spine in scoliosis is in a coil formation that not only curves side to side, but also twists forward and back. If all you are trying to do is bend it from side to side, you are never going to be able to effectively straighten it unless you also address the spiral bend in the coil first.
Because the scoliosis curvature is simply part of a global neurological pattern that affects the entire body, attempts to just "fix" local-level curvatures are doomed to achieve only short-lived results. This larger neurological element must be properly addressed if you expect to halt the progression of a scoliosis curve, especially in the rapid growth phase just before puberty.
A final analogy that's important to consider when managing scoliosis patients: Think of the scoliosis spine as a wet washcloth that is being wrung out. As it twists, it shortens and buckles on itself. The spine acts the same way. Each vertebral segment tends to lock out in 4 degrees of rotation. So, to straighten out the lateral curve, you first have to simultaneously "unwring" it by releasing the shortened and restricted rotational muscles of the spine while simultaneously unloading the spine axially. Once you have corrected this rotational dysfunction and strengthened the appropriate antagonist muscle, the lateral deformity caused by the collapsing of the spine will improve and you will experience greater success and more lasting results with your scoliosis patients.
Scoliosis on Two-Dimensional X-Ray – A Limited View


Although a two-dimensional radiograph will quite readily reveal the lateral curves of scoliosis, because it is not a three-dimensional representation of the body it does nothing to reveal the all-important rotational component most often found in scoliosis. One of the greatest challenges in treating scoliosis is for the practitioner to truly appreciate the full depth of the problem. This difficulty lies greatly in the limitations of the primary diagnostic tool used for scoliosis, the plain-film X-ray. Although X-rays of a scoliosis are quite dramatic, they are still just a two-dimensional representation of an even more complex three-dimensional problem. Thus, simply viewing X-rays of a scoliosis often draws the attention of the doctor to only the two obvious dimensions of the spine, the vertical height and the lateral deviation. This focus results in significant visual limitations for doctor because it fails to capture the third dimension of the scoliosis.
The difference in the level of conceptual understanding of scoliosis of the spine can best be illustrated by considering the differences between a two- and three-dimensional chess board. Although the classical game of chess is played in only two dimensions, it is still an intellectually challenging game that requires a great deal of global thinking to strategize how the various combinations of pieces interact on the board. Now imagine just how much more thinking is required in a game of chess on a three-dimensional board (for example, as portrayed on many episodes of "Star Trek"). Instead of just thinking about moving pieces in two planes, consider attacks and counterattacks in a third dimension. This adds a whole other level of complexity to your play. The same increased level of conceptual thinking is required to effectively treat a three-dimensional scoliosis.
Appreciating the Unseen Core Element of Scoliosis
When they are viewed on X-ray, scoliosis curves appear "S" shaped and span both the thoracic and lumbar spine; however, the reality is that there is far more to this curve than just meets the eye. Because X-rays are only two-dimensional representations of a three-dimensional body, what looks like a flattened-out "S" curve on an X-ray is in reality more like the helix of a spring in the body. Understanding this distinction is critical when treating the spine to stop the progression of or correct a scoliosis curvature.
This helix-shaped curvature is caused by a unilateral rotation dysfunction of the deep postural muscles surrounding the spine. There is a neurological basis to this imbalance and it is at the heart of the scoliosis. Correction and stabilization of these muscular imbalances must be addressed in order to stop progression of the curve.
Unfortunately, this three-dimensional rotational twist is often overlooked during the treatment process, which can cause dire consequences. When a chiropractor does not take this rotational component into consideration and applies nonspecific bilateral treatment to the spine, they can actually cause an increase in the curvature by contributing to the unilateral rotation dysfunction; therefore, this type of generalized adjusting is contraindicated for scoliosis.
While a rotatory force applied in the opposite direction of the torque will help correct a unilateral rotation dysfunction, doing adjustments in the direction of the scoliosis spiral will actually serve to reinforce the abnormal movement patterns and end up worsening the condition. Additionally, adjustments that are aimed solely on straightening the concave side of the curve in an attempt to reduce a two-dimensional Cobb angle on X-ray can also be detrimental if the aforementioned rotation component is not taken into consideration.
Take a Global Approach to Help Your Scoliosis Patients
Another analogy to help illustrate the inadequacy of this two-dimensional approach is to think of trying to straighten out a metal spring. Much like a spring, the spine in scoliosis is in a coil formation that not only curves side to side, but also twists forward and back. If all you are trying to do is bend it from side to side, you are never going to be able to effectively straighten it unless you also address the spiral bend in the coil first.
Because the scoliosis curvature is simply part of a global neurological pattern that affects the entire body, attempts to just "fix" local-level curvatures are doomed to achieve only short-lived results. This larger neurological element must be properly addressed if you expect to halt the progression of a scoliosis curve, especially in the rapid growth phase just before puberty.
A final analogy that's important to consider when managing scoliosis patients: Think of the scoliosis spine as a wet washcloth that is being wrung out. As it twists, it shortens and buckles on itself. The spine acts the same way. Each vertebral segment tends to lock out in 4 degrees of rotation. So, to straighten out the lateral curve, you first have to simultaneously "unwring" it by releasing the shortened and restricted rotational muscles of the spine while simultaneously unloading the spine axially. Once you have corrected this rotational dysfunction and strengthened the appropriate antagonist muscle, the lateral deformity caused by the collapsing of the spine will improve and you will experience greater success and more lasting results with your scoliosis patients.

Dr. Brett Diaz is a 1987 graduate of Los Angeles College of Chiropractic (now Southern California University of Health Sciences). He is the director of clinical services for scoliosis treatment alternatives at O.C. Back & Body Doctors, in Irvine, Calif., where he specializes in nonsurgical treatment of scoliosis utilizing a variety of conservative modalities including chiropractic care, physical therapy, massage therapy, dynamic bracing, vibration therapy, scoliosis-specific exercises, active release techniques, Pilates, and de-rotation traction therapy. 

Dr. Brett Diaz is a 1987 graduate of Los Angeles College of Chiropractic (now Southern California University of Health Sciences). He is the director of clinical services for scoliosis treatment alternatives at O.C. Back & Body Doctors, in Irvine, Calif., where he specializes in nonsurgical treatment of scoliosis utilizing a variety of conservative modalities including chiropractic care, physical therapy, massage therapy, dynamic bracing, vibration therapy, scoliosis-specific exercises, active release techniques, Pilates, and de-rotation traction therapy. 
Read More


The Cast Curse

By LCC-Catie D, Sunday at 8:30 am



The Cast Curse
Our little Van Gogh's self portrait
Hopefully you survived the first post and have agreed to stay with us for more Suck It, Scoliosis.  You learned from meeting our curvy crew we chose casting as our treatment modality.  Although this method of treatment is the best course of action for infants and toddlers, it is not as widely used for children with juvenile scoliosis.  The science behind casting infants and toddlers is to harness their rapid growth to use as a corrective force in straightening their little spines.  Scoliosis was once thought to be a 2 dimensional curve of the spine.  Through great research and study it has actually been determined to be  3 dimensional with both a curve and rotation of the spine.  With casting, our great surgeon at Chicago Shriner's Hospital for Children is able to place our Iron Will under general anesthesia, place him in full traction where he manipulates his rotation and curve straight and applies a plaster cast  wrapped in fiberglass to his torso to allow him to safely grow with his spine in this straight position.  Iron Will wears his armor for 10-12 weeks and then we go at it once again.    OMG that was way too technical! I was afraid if I told you they knock him out, place him in a medieval torcher device, beat the crap out of his little body then wrap him in plaster and fiberglass I may have lost you.  It definitely is not the vision you want to have of your son but it gives him a fighting chance and at this point we will take a stinky cast kid over the alternative.


Cast #1 came and went with little drama; unless you count learning to crawl and walk while wearing a plaster cast half your body weight dramatic.  To be honest 10 casts and 2 years have passed so those battles have faded into something we did along the way.  The one aspect of casting I will never be able to erase is the curse.  Any parent who has lived it or is living it will tell you the curse will take you on a ride you will never forget. 

At first I thought he must have a stomach virus.  Daycare was notorious for sending home these little blessings.  After cast #2, 3, 4 & 5 all had the same symptoms I started to rethink what was going on.  Some families have problems with vomiting after anesthesia.  We were fortunate not to have this as one of Iron Will's  side effects.  But somehow the mysterious vomiting would appear after about 3-4 days at home. 
            Our first visit from the vomit fairy was enough to keep you on your toes.  Vomit soaked in cotton and plaster is some nasty shit you will never forget.  After cast number 2, I was taking my usual Friday trip to drop my oldest son at school.  I must warn you I am the mom who drops off at school in her Pj's, no bra and houseshoes.  It is not a pretty picture people.  Iron WIll had shown no signs he wasn’t feeling well and until I heard the familiar Blah and scream from my other darling I had no reason to suspect anything was wrong.  As I pulled into the Ring around the Rosary circle, I panicked at what to do.  Iron Will, the cast, his carseat and half my backseat were covered in vomit.  I instructed a stunned older child to run into school and ask my sister, the teacher,  for something so I could clean William up.  A few minutes later came out to the van, still parked in the circle, with several paint shirts and paper towels.  I kissed him and told him 'we would be fine' & pulled around the circle and into a parking spot where I began the clean up for the 20 minute ride home.  Me in my pajama's sans bra  in the parking lot of the Catholic school I pulled Iron Will out of his seat and tried my hardest to take off his jammies and put him in the front seat of the van as I scooped vomit out of his seat.  Thank the lord above I use my mommi-van to hoard all sorts of random crap.  I spent 10 minutes in the parking lot of the school until I felt I could buckle him in the seat and proceed on our merry way.  About 10 minutes into the drive I heard the all to familiar sound again.  Ding Ding Ding round two of vomit in the car.  There was no sense in pulling over at this point, buckle down and get home.  I arrived at home and took William into the house.  There was vomit everywhere.  The smell is still something I can conjour by just writing this horrid tale.  I used a warm cloth and cleansed the outside of the cast as best I could.  For the nasty matter which made it down the front of the cast, I soaked a panty hose in alcohol and fished it through the cast.  As I was flossing, nasty ass chunks of breakfast past  came shooting out the bottom.  This fricking thing is never going to be the same.  I am not going to be the parent to call Linda at Chicago Shriners and tell he their work of art has been ruined.  This was as clean as he was going to get.  I put a new sleeper on, placed a bib over the top and put him in bed. We had repeat vomiting off and on for about a week before things returned to "normal." 
            What we believed to be random on cast number two proved clockwork on cast three and four. For cast number three it came in the middle of the night approximately 5 days after initial casting.  This little jewel lasted approximately 3 days and disappeared as mysteriously as it arrived.  For cast five, it came during a family meal at a local Bar B Q restaurant.  Dinner was finished and I let Mammaw/Pappaw enjoy the boys so I could finally pee in peace.  I wasn’t in there more than a  minute when I heard William start to cry and tables moving.  I washed my hands and opened the door.  Holy crap, my father in law was holding William with vomit all over his back. My hubby and the owner of the restaurant were standing beside him with paper towel and the large kitchen trash can.  William was still crying and vomiting.  I seriously considered closing the door and pretending non of this was happening.  My older son upon seeing me  of course pipes up with 'Mom it’s the curse'!  At this point I was pretty sure he was correct, it was the curse.  Needless to say we exited stage left as quickly as possible.  I ran to the van with William, stripped off the old clothes cleaned the cast with wipes and put him in a sleeper I had in his diaper bag.  Before I could get him buckled into his seat and hubby to the van, it hit again.  This time I was victim along with the only sleeper we had.  I strapped a naked jay bird and a tiny feeding bib back in the carseat and thought fudge, here we go again.  My father in law is a good fricken sport.  He tossed his puke shirt in the bathroom trash and left a big ass tip! We fled the seen never to show our faces again. 
 The curse came and went each cast until cast #6 when i was waiting for the shoe to drop and it didn't.  As with any curse I really don't know if it is real or simply a test Scoliosis sent to make us second guess our decision to cast our child.  The fact we survived the curse and went on to cast again is a SUCK It, Scoliosis moment.   Until next time, Keep it Curvy!
Catie(Scoliosis Sucks) D.
Stay with us for our next post: My First Dear John letter...

About Dr Kevin Lau
Dr Kevin Lau DC is the founder of Health In Your Hands, a series of tools for Scoliosis prevention and treatment. The set includes his book Your Plan for Natural Scoliosis Prevention and Treatment, a companion Scoliosis Exercises for Prevention and Correction DVD and the innovative new iPhone application ScolioTrack. Dr Kevin Lau D.C. is a graduate in Doctor of Chiropractic from RMIT University in Melbourne Australia and Masters in Holistic Nutrition from Clayton College of Natural Health in USA. In 2006 I was awarded the "Best Health-care Provider Awards" by the largest Newspaper publication in Singapore on October 18 2006 as well as being interviewed on Primetime Channel News Asia as well as other TV and Radio. For more information on Dr Kevin Lau, watch his interviews or get a free sneak peek of his book, go to: http://www.hiyh.info.
By LCC-Catie D, Sunday at 8:30 am



The Cast Curse
Our little Van Gogh's self portrait
Hopefully you survived the first post and have agreed to stay with us for more Suck It, Scoliosis.  You learned from meeting our curvy crew we chose casting as our treatment modality.  Although this method of treatment is the best course of action for infants and toddlers, it is not as widely used for children with juvenile scoliosis.  The science behind casting infants and toddlers is to harness their rapid growth to use as a corrective force in straightening their little spines.  Scoliosis was once thought to be a 2 dimensional curve of the spine.  Through great research and study it has actually been determined to be  3 dimensional with both a curve and rotation of the spine.  With casting, our great surgeon at Chicago Shriner's Hospital for Children is able to place our Iron Will under general anesthesia, place him in full traction where he manipulates his rotation and curve straight and applies a plaster cast  wrapped in fiberglass to his torso to allow him to safely grow with his spine in this straight position.  Iron Will wears his armor for 10-12 weeks and then we go at it once again.    OMG that was way too technical! I was afraid if I told you they knock him out, place him in a medieval torcher device, beat the crap out of his little body then wrap him in plaster and fiberglass I may have lost you.  It definitely is not the vision you want to have of your son but it gives him a fighting chance and at this point we will take a stinky cast kid over the alternative.


Cast #1 came and went with little drama; unless you count learning to crawl and walk while wearing a plaster cast half your body weight dramatic.  To be honest 10 casts and 2 years have passed so those battles have faded into something we did along the way.  The one aspect of casting I will never be able to erase is the curse.  Any parent who has lived it or is living it will tell you the curse will take you on a ride you will never forget. 

At first I thought he must have a stomach virus.  Daycare was notorious for sending home these little blessings.  After cast #2, 3, 4 & 5 all had the same symptoms I started to rethink what was going on.  Some families have problems with vomiting after anesthesia.  We were fortunate not to have this as one of Iron Will's  side effects.  But somehow the mysterious vomiting would appear after about 3-4 days at home. 
            Our first visit from the vomit fairy was enough to keep you on your toes.  Vomit soaked in cotton and plaster is some nasty shit you will never forget.  After cast number 2, I was taking my usual Friday trip to drop my oldest son at school.  I must warn you I am the mom who drops off at school in her Pj's, no bra and houseshoes.  It is not a pretty picture people.  Iron WIll had shown no signs he wasn’t feeling well and until I heard the familiar Blah and scream from my other darling I had no reason to suspect anything was wrong.  As I pulled into the Ring around the Rosary circle, I panicked at what to do.  Iron Will, the cast, his carseat and half my backseat were covered in vomit.  I instructed a stunned older child to run into school and ask my sister, the teacher,  for something so I could clean William up.  A few minutes later came out to the van, still parked in the circle, with several paint shirts and paper towels.  I kissed him and told him 'we would be fine' & pulled around the circle and into a parking spot where I began the clean up for the 20 minute ride home.  Me in my pajama's sans bra  in the parking lot of the Catholic school I pulled Iron Will out of his seat and tried my hardest to take off his jammies and put him in the front seat of the van as I scooped vomit out of his seat.  Thank the lord above I use my mommi-van to hoard all sorts of random crap.  I spent 10 minutes in the parking lot of the school until I felt I could buckle him in the seat and proceed on our merry way.  About 10 minutes into the drive I heard the all to familiar sound again.  Ding Ding Ding round two of vomit in the car.  There was no sense in pulling over at this point, buckle down and get home.  I arrived at home and took William into the house.  There was vomit everywhere.  The smell is still something I can conjour by just writing this horrid tale.  I used a warm cloth and cleansed the outside of the cast as best I could.  For the nasty matter which made it down the front of the cast, I soaked a panty hose in alcohol and fished it through the cast.  As I was flossing, nasty ass chunks of breakfast past  came shooting out the bottom.  This fricking thing is never going to be the same.  I am not going to be the parent to call Linda at Chicago Shriners and tell he their work of art has been ruined.  This was as clean as he was going to get.  I put a new sleeper on, placed a bib over the top and put him in bed. We had repeat vomiting off and on for about a week before things returned to "normal." 
            What we believed to be random on cast number two proved clockwork on cast three and four. For cast number three it came in the middle of the night approximately 5 days after initial casting.  This little jewel lasted approximately 3 days and disappeared as mysteriously as it arrived.  For cast five, it came during a family meal at a local Bar B Q restaurant.  Dinner was finished and I let Mammaw/Pappaw enjoy the boys so I could finally pee in peace.  I wasn’t in there more than a  minute when I heard William start to cry and tables moving.  I washed my hands and opened the door.  Holy crap, my father in law was holding William with vomit all over his back. My hubby and the owner of the restaurant were standing beside him with paper towel and the large kitchen trash can.  William was still crying and vomiting.  I seriously considered closing the door and pretending non of this was happening.  My older son upon seeing me  of course pipes up with 'Mom it’s the curse'!  At this point I was pretty sure he was correct, it was the curse.  Needless to say we exited stage left as quickly as possible.  I ran to the van with William, stripped off the old clothes cleaned the cast with wipes and put him in a sleeper I had in his diaper bag.  Before I could get him buckled into his seat and hubby to the van, it hit again.  This time I was victim along with the only sleeper we had.  I strapped a naked jay bird and a tiny feeding bib back in the carseat and thought fudge, here we go again.  My father in law is a good fricken sport.  He tossed his puke shirt in the bathroom trash and left a big ass tip! We fled the seen never to show our faces again. 
 The curse came and went each cast until cast #6 when i was waiting for the shoe to drop and it didn't.  As with any curse I really don't know if it is real or simply a test Scoliosis sent to make us second guess our decision to cast our child.  The fact we survived the curse and went on to cast again is a SUCK It, Scoliosis moment.   Until next time, Keep it Curvy!
Catie(Scoliosis Sucks) D.
Stay with us for our next post: My First Dear John letter...

About Dr Kevin Lau
Dr Kevin Lau DC is the founder of Health In Your Hands, a series of tools for Scoliosis prevention and treatment. The set includes his book Your Plan for Natural Scoliosis Prevention and Treatment, a companion Scoliosis Exercises for Prevention and Correction DVD and the innovative new iPhone application ScolioTrack. Dr Kevin Lau D.C. is a graduate in Doctor of Chiropractic from RMIT University in Melbourne Australia and Masters in Holistic Nutrition from Clayton College of Natural Health in USA. In 2006 I was awarded the "Best Health-care Provider Awards" by the largest Newspaper publication in Singapore on October 18 2006 as well as being interviewed on Primetime Channel News Asia as well as other TV and Radio. For more information on Dr Kevin Lau, watch his interviews or get a free sneak peek of his book, go to: http://www.hiyh.info.
Read More


5 Foods That Can Trigger a Stroke

Crackers, chips, and store-bought pastries and baked goods
Muffins, doughnuts, chips, crackers, and many other baked goods are high in trans fats, which are hydrogenated oils popular with commercial bakeries because they stay solid at room temperature, so the products don't require refrigeration. Also listed on labels as "partially hydrogenated" or hydrogenated oils, trans fats are found in all kinds of snack foods, frozen foods, and baked goods, including salad dressings, microwave popcorn, stuffing mixes, frozen tater tots and French fries, cake mixes, and whipped toppings. They're also what makes margarine stay in a solid cube. The worst offenders are fried fast foods such as onion rings, French fries, and fried chicken.

Why it's bad
For years scientists have known trans fats are dangerous artery-blockers, upping the concentrations of lipids and bad cholesterol in the blood and lowering good cholesterol. Now we can add stroke to the list of dangers. This year researchers at the University of North Carolina found that women who ate 7 grams of trans fat each day -- about the amount in two doughnuts or half a serving of French fries -- had 30 percent more strokes (the ischemic type, caused by blocked blood flow to the brain) than women who ate just 1 gram a day. Another recent study, also in women, found that trans fats promoted inflammation and higher levels of C-reactive protein, which have been linked to an increased risk of diabetes, heart disease, and stroke.



What to do
Aim to limit trans fats to no more than 1 or 2 grams a day -- and preferably none. Avoid fast-food French fries and other fried menu items and study packaged food labels closely. Even better, bake your own cookies, cakes, and other snacks. When you can't, search out "health-food" alternative snacks, such as Terra brand potato chips and traditional whole grain crackers such as those made by Finn, Wasa, AkMak, Ryvita, and Lavasch.

Smoked and processed meats

Whether your weakness is pastrami, sausage, hot dogs, bacon, or a smoked turkey sandwich, the word from the experts is: Watch out.

Why it's bad
Smoked and processed meats are nasty contributors to stroke risk in two ways: The preserving processes leave them packed with sodium, but even worse are the preservatives used to keep processed meats from going bad. Sodium nitrate and nitrite have been shown by researchers to directly damage blood vessels, causing arteries to harden and narrow. And of course damaged, overly narrow blood vessels are exactly what you don't want if you fear stroke.

Many studies have linked processed meats to coronary artery disease (CAD); one meta-analysis in the journal Circulation calculated a 42-percent increase in coronary heart disease for those who eat one serving of processed meat a day. Stroke is not the only concern for salami fans; cancer journals have reported numerous studies in the past few years showing that consumption of cured and smoked meats is linked with increased risk of diabetes and higher incidences of numerous types of cancer, including leukemia.

What to do
If a smoked turkey or ham sandwich is your lunch of choice, try to vary your diet, switching to tuna, peanut butter, or other choices several days a week. Or cook turkey and chicken yourself and slice it thin for sandwiches

Diet soda

Although replacing sugary drinks with diet soda seems like a smart solution for keeping weight down -- a heart-healthy goal -- it turns out diet soda is likely a major bad guy when it comes to stroke.

Why it's bad
People who drink a diet soda a day may up their stroke risk by 48 percent. A Columbia University study presented at the American Stroke Association's 2011 International Stroke Conference followed 2,500 people ages 40 and older and found that daily diet soda drinkers had 60 percent more strokes, heart attacks, and coronary artery disease than those who didn't drink diet soda. Researchers don't know exactly how diet soda ups stroke risk -- and are following up with further studies -- but nutritionists are cautioning anyone concerned about stroke to cut out diet soda pop.

What to do
Substitute more water for soda in your daily diet. It's the healthiest thirst-quencher by far, researchers say. If you don't like water, try lemonade, iced tea, or juice.
Red meat

This winter, when the respected journal Stroke published a study showing that women who consumed a large portion of red meat each day had a 42-percent higher incidence of stroke, it got nutrition experts talking. The information that red meat, with its high saturated fat content, isn't healthy for those looking to prevent heart disease and stroke wasn't exactly news. But the percentage increase (almost 50 percent!) was both startling and solid; the researchers arrived at their finding after following 35,000 Swedish women for ten years.

Why it's bad
Researchers have long known that the saturated fat in red meat raises the risk of stroke and heart disease by gradually clogging arteries with a buildup of protein plaques. Now it turns out that hemoglobin, the ingredient that gives red meat its high iron content, may pose a specific danger when it comes to stroke. Researchers are investigating whether blood becomes thicker and more viscous as a result of the consumption of so-called heme iron, specifically upping the chance of strokes.

What to do
Aim to substitute more poultry -- particularly white meat -- and fish, which are low in heme iron, for red meat. Also, choose the heart-healthiest sources of protein whenever you can, especially beans, legumes, nuts, tofu, and nonfat dairy.

Canned soup and prepared foods

Whether it's canned soup, canned spaghetti, or healthy-sounding frozen dinners, prepared foods and mixes rely on sodium to increase flavor and make processed foods taste fresher. Canned soup is cited by nutritionists as the worst offender; one can of canned chicken noodle soup contains more than 1,100 mg of sodium, while many other varieties, from clam chowder to simple tomato, have between 450 and 800 mg per serving. Compare that to the American Heart and Stroke Association's recommendation of less than1,500 mg of sodium daily and you'll see the problem. In fact, a nutritionist-led campaign, the National Salt Reduction Initiative, calls on food companies to reduce the salt content in canned soup and other products by 20 percent in the next two years.

Why it's bad
Salt, or sodium as it's called on food labels, directly affects stroke risk. In one recent study, people who consumed more than 4,000 mg of sodium daily had more than double the risk of stroke compared to those who ate 2,000 mg or less. Yet the Centers for Disease Control estimate that most Americans eat close to 3,500 mg of sodium per day. Studies show that sodium raises blood pressure, the primary causative factor for stroke. And be warned: Sodium wears many tricky disguises, which allow it to hide in all sorts of foods that we don't necessarily think of as salty. Some common, safe-sounding ingredients that really mean salt:


Baking soda

Baking powder

MSG (monosodium glutamate)

Disodium phosphate

Sodium alginate

What to do
Make your own homemade soups and entrees, then freeze individual serving-sized portions. Buy low-sodium varieties, but read labels carefully, since not all products marked "low sodium" live up to that promise.
Crackers, chips, and store-bought pastries and baked goods
Muffins, doughnuts, chips, crackers, and many other baked goods are high in trans fats, which are hydrogenated oils popular with commercial bakeries because they stay solid at room temperature, so the products don't require refrigeration. Also listed on labels as "partially hydrogenated" or hydrogenated oils, trans fats are found in all kinds of snack foods, frozen foods, and baked goods, including salad dressings, microwave popcorn, stuffing mixes, frozen tater tots and French fries, cake mixes, and whipped toppings. They're also what makes margarine stay in a solid cube. The worst offenders are fried fast foods such as onion rings, French fries, and fried chicken.

Why it's bad
For years scientists have known trans fats are dangerous artery-blockers, upping the concentrations of lipids and bad cholesterol in the blood and lowering good cholesterol. Now we can add stroke to the list of dangers. This year researchers at the University of North Carolina found that women who ate 7 grams of trans fat each day -- about the amount in two doughnuts or half a serving of French fries -- had 30 percent more strokes (the ischemic type, caused by blocked blood flow to the brain) than women who ate just 1 gram a day. Another recent study, also in women, found that trans fats promoted inflammation and higher levels of C-reactive protein, which have been linked to an increased risk of diabetes, heart disease, and stroke.



What to do
Aim to limit trans fats to no more than 1 or 2 grams a day -- and preferably none. Avoid fast-food French fries and other fried menu items and study packaged food labels closely. Even better, bake your own cookies, cakes, and other snacks. When you can't, search out "health-food" alternative snacks, such as Terra brand potato chips and traditional whole grain crackers such as those made by Finn, Wasa, AkMak, Ryvita, and Lavasch.

Smoked and processed meats

Whether your weakness is pastrami, sausage, hot dogs, bacon, or a smoked turkey sandwich, the word from the experts is: Watch out.

Why it's bad
Smoked and processed meats are nasty contributors to stroke risk in two ways: The preserving processes leave them packed with sodium, but even worse are the preservatives used to keep processed meats from going bad. Sodium nitrate and nitrite have been shown by researchers to directly damage blood vessels, causing arteries to harden and narrow. And of course damaged, overly narrow blood vessels are exactly what you don't want if you fear stroke.

Many studies have linked processed meats to coronary artery disease (CAD); one meta-analysis in the journal Circulation calculated a 42-percent increase in coronary heart disease for those who eat one serving of processed meat a day. Stroke is not the only concern for salami fans; cancer journals have reported numerous studies in the past few years showing that consumption of cured and smoked meats is linked with increased risk of diabetes and higher incidences of numerous types of cancer, including leukemia.

What to do
If a smoked turkey or ham sandwich is your lunch of choice, try to vary your diet, switching to tuna, peanut butter, or other choices several days a week. Or cook turkey and chicken yourself and slice it thin for sandwiches

Diet soda

Although replacing sugary drinks with diet soda seems like a smart solution for keeping weight down -- a heart-healthy goal -- it turns out diet soda is likely a major bad guy when it comes to stroke.

Why it's bad
People who drink a diet soda a day may up their stroke risk by 48 percent. A Columbia University study presented at the American Stroke Association's 2011 International Stroke Conference followed 2,500 people ages 40 and older and found that daily diet soda drinkers had 60 percent more strokes, heart attacks, and coronary artery disease than those who didn't drink diet soda. Researchers don't know exactly how diet soda ups stroke risk -- and are following up with further studies -- but nutritionists are cautioning anyone concerned about stroke to cut out diet soda pop.

What to do
Substitute more water for soda in your daily diet. It's the healthiest thirst-quencher by far, researchers say. If you don't like water, try lemonade, iced tea, or juice.
Red meat

This winter, when the respected journal Stroke published a study showing that women who consumed a large portion of red meat each day had a 42-percent higher incidence of stroke, it got nutrition experts talking. The information that red meat, with its high saturated fat content, isn't healthy for those looking to prevent heart disease and stroke wasn't exactly news. But the percentage increase (almost 50 percent!) was both startling and solid; the researchers arrived at their finding after following 35,000 Swedish women for ten years.

Why it's bad
Researchers have long known that the saturated fat in red meat raises the risk of stroke and heart disease by gradually clogging arteries with a buildup of protein plaques. Now it turns out that hemoglobin, the ingredient that gives red meat its high iron content, may pose a specific danger when it comes to stroke. Researchers are investigating whether blood becomes thicker and more viscous as a result of the consumption of so-called heme iron, specifically upping the chance of strokes.

What to do
Aim to substitute more poultry -- particularly white meat -- and fish, which are low in heme iron, for red meat. Also, choose the heart-healthiest sources of protein whenever you can, especially beans, legumes, nuts, tofu, and nonfat dairy.

Canned soup and prepared foods

Whether it's canned soup, canned spaghetti, or healthy-sounding frozen dinners, prepared foods and mixes rely on sodium to increase flavor and make processed foods taste fresher. Canned soup is cited by nutritionists as the worst offender; one can of canned chicken noodle soup contains more than 1,100 mg of sodium, while many other varieties, from clam chowder to simple tomato, have between 450 and 800 mg per serving. Compare that to the American Heart and Stroke Association's recommendation of less than1,500 mg of sodium daily and you'll see the problem. In fact, a nutritionist-led campaign, the National Salt Reduction Initiative, calls on food companies to reduce the salt content in canned soup and other products by 20 percent in the next two years.

Why it's bad
Salt, or sodium as it's called on food labels, directly affects stroke risk. In one recent study, people who consumed more than 4,000 mg of sodium daily had more than double the risk of stroke compared to those who ate 2,000 mg or less. Yet the Centers for Disease Control estimate that most Americans eat close to 3,500 mg of sodium per day. Studies show that sodium raises blood pressure, the primary causative factor for stroke. And be warned: Sodium wears many tricky disguises, which allow it to hide in all sorts of foods that we don't necessarily think of as salty. Some common, safe-sounding ingredients that really mean salt:


Baking soda

Baking powder

MSG (monosodium glutamate)

Disodium phosphate

Sodium alginate

What to do
Make your own homemade soups and entrees, then freeze individual serving-sized portions. Buy low-sodium varieties, but read labels carefully, since not all products marked "low sodium" live up to that promise.
Read More


Sunday, October 2, 2011

Vitamin K2 Supplementation Improves Insulin Sensitivity via Osteocalcin Metabolism: A Placebo-Controlled Trial


Research is showing that vitamin K can help to regulate your glucose metabolism.  This may be because the vitamin converts to a substance called carboxylated osteocalcin in your body, which affects insulin sensitivity.
A recent study found that that vitamin K2 supplementation increased insulin sensitivity in healthy young men.  The effect seemed to be related to increased carboxylated osteocalcin levels, rather than to another factor such as modulation of inflammation.
According to the study in Diabetes Care:



Undercarboxylated osteocalcin (ucOC) is reported to function as an endocrine hormone, affecting glucose metabolism in mice (1,2). Vitamin K, which converts ucOC to carboxylated osteocalcin (cOC), has been suggested to regulate glucose metabolism by modulating osteocalcin and/or proinflammatory pathway (3–5).

We studied whether modulation of ucOC via vitamin K2 supplementation for 4 weeks affects β-cell function and/or insulin sensitivity in healthy young male subjects. Forty-two healthy young male volunteers received vitamin K2 (menatetrenone; 30 mg; Eisai Co., Japan) or placebo t.i.d. for 4 weeks. Frequently sampled intravenous glucose tolerance test was performed to determine insulin sensitivity index (Si), acute insulin response to glucose (AIRg), and disposition index (DI) before and after treatment. Adiponectin, interleukin (IL)-6, C-reactive protein (CRP), ucOC, and cOC were measured before and after treatment.

After excluding frequently sampled intravenous glucose tolerance test failures (n = 4) and extreme outliers (n = 5), 18 subjects in the treatment group and 15 subjects in the control group were finally analyzed. The institutional review board of Seoul National University Hospital approved this study.

The age (29 [24–31] vs. 29 [25.5–31.5] years, median [interquartile range]) and BMI (24.9 [22.9–26.8] vs. 25.3 [21.9–27.0] kg/cm2) of the control and treatment groups were not significantly different. Vitamin K2 supplementation significantly increased Si (4.4 [3.2–5.6] vs. 6.6 [4.3–9.6]; P = 0.01) and DI (2,266 [1,536–2,785] vs. 3,025 [2,441–4,835]; P < 0.01), but these indices were not affected by placebo treatment. The percent increase in DI was significantly higher in the vitamin K2 group compared with the placebo group (50.9 [20.8–87.3] vs. 2.7 [-10.0 to 39.2]%; P = 0.03) resulting in higher posttreatment DI (3,025 [2,441–4,835] vs. 1,838 [1,320–2,741]; P = 0.01). These differences persisted even after adjusting for baseline Si, AIRg, DI, ucOC, cOC, IL-6, adiponectin, and CRP (P = 0.008 for percent increase in DI and P = 0.001 for posttreatment DI). Treatment with vitamin K2 decreased ucOC (0.9 [0.5–1.8] vs. 0.4 [0.4–0.6] ng/mL; P = 0.02) and increased cOC (9.6 [7.1–15.1] vs. 16.0 [12.4–16.0] ng/mL; P = 0.01). However, no significant changes were observed in AIRg, fasting plasma glucose, weight, IL-6, CRP, or adiponectin (data not shown). Si fold change was significantly associated with baseline cOC, baseline ucOC, and cOC fold change even after adjusting for age and weight fold change (all P < 0.05).

To summarize, we have demonstrated for the first time that vitamin K2 supplementation for 4 weeks increased insulin sensitivity in healthy young men, which seems to be related to increased cOC rather than modulation of inflammation. Small sample size limits firm interpretation on β-cell function. Our results are consistent with previous studies that demonstrated improved glucose intolerance or relieved insulin resistance by treatment with vitamin K1 (3) or vitamin K2 (4), respectively. We conclude that unlike in rodents, cOC rather than ucOC may be the endocrine hormone that increases insulin sensitivity in humans. Although our study could not provide the underlying mechanism, we speculate that cOC or vitamin K could modulate adipokines or inflammatory pathways other than the IL-6 pathways. Alternatively, cOC can directly regulate glucose disposal at skeletal muscle or adipose tissues. Further studies to elucidate the mechanism of action are warranted.

Research is showing that vitamin K can help to regulate your glucose metabolism.  This may be because the vitamin converts to a substance called carboxylated osteocalcin in your body, which affects insulin sensitivity.
A recent study found that that vitamin K2 supplementation increased insulin sensitivity in healthy young men.  The effect seemed to be related to increased carboxylated osteocalcin levels, rather than to another factor such as modulation of inflammation.
According to the study in Diabetes Care:



Undercarboxylated osteocalcin (ucOC) is reported to function as an endocrine hormone, affecting glucose metabolism in mice (1,2). Vitamin K, which converts ucOC to carboxylated osteocalcin (cOC), has been suggested to regulate glucose metabolism by modulating osteocalcin and/or proinflammatory pathway (3–5).

We studied whether modulation of ucOC via vitamin K2 supplementation for 4 weeks affects β-cell function and/or insulin sensitivity in healthy young male subjects. Forty-two healthy young male volunteers received vitamin K2 (menatetrenone; 30 mg; Eisai Co., Japan) or placebo t.i.d. for 4 weeks. Frequently sampled intravenous glucose tolerance test was performed to determine insulin sensitivity index (Si), acute insulin response to glucose (AIRg), and disposition index (DI) before and after treatment. Adiponectin, interleukin (IL)-6, C-reactive protein (CRP), ucOC, and cOC were measured before and after treatment.

After excluding frequently sampled intravenous glucose tolerance test failures (n = 4) and extreme outliers (n = 5), 18 subjects in the treatment group and 15 subjects in the control group were finally analyzed. The institutional review board of Seoul National University Hospital approved this study.

The age (29 [24–31] vs. 29 [25.5–31.5] years, median [interquartile range]) and BMI (24.9 [22.9–26.8] vs. 25.3 [21.9–27.0] kg/cm2) of the control and treatment groups were not significantly different. Vitamin K2 supplementation significantly increased Si (4.4 [3.2–5.6] vs. 6.6 [4.3–9.6]; P = 0.01) and DI (2,266 [1,536–2,785] vs. 3,025 [2,441–4,835]; P < 0.01), but these indices were not affected by placebo treatment. The percent increase in DI was significantly higher in the vitamin K2 group compared with the placebo group (50.9 [20.8–87.3] vs. 2.7 [-10.0 to 39.2]%; P = 0.03) resulting in higher posttreatment DI (3,025 [2,441–4,835] vs. 1,838 [1,320–2,741]; P = 0.01). These differences persisted even after adjusting for baseline Si, AIRg, DI, ucOC, cOC, IL-6, adiponectin, and CRP (P = 0.008 for percent increase in DI and P = 0.001 for posttreatment DI). Treatment with vitamin K2 decreased ucOC (0.9 [0.5–1.8] vs. 0.4 [0.4–0.6] ng/mL; P = 0.02) and increased cOC (9.6 [7.1–15.1] vs. 16.0 [12.4–16.0] ng/mL; P = 0.01). However, no significant changes were observed in AIRg, fasting plasma glucose, weight, IL-6, CRP, or adiponectin (data not shown). Si fold change was significantly associated with baseline cOC, baseline ucOC, and cOC fold change even after adjusting for age and weight fold change (all P < 0.05).

To summarize, we have demonstrated for the first time that vitamin K2 supplementation for 4 weeks increased insulin sensitivity in healthy young men, which seems to be related to increased cOC rather than modulation of inflammation. Small sample size limits firm interpretation on β-cell function. Our results are consistent with previous studies that demonstrated improved glucose intolerance or relieved insulin resistance by treatment with vitamin K1 (3) or vitamin K2 (4), respectively. We conclude that unlike in rodents, cOC rather than ucOC may be the endocrine hormone that increases insulin sensitivity in humans. Although our study could not provide the underlying mechanism, we speculate that cOC or vitamin K could modulate adipokines or inflammatory pathways other than the IL-6 pathways. Alternatively, cOC can directly regulate glucose disposal at skeletal muscle or adipose tissues. Further studies to elucidate the mechanism of action are warranted.
Read More