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Saturday, March 26, 2011

Is Trading "Deformity for Dysfunction" a Good Deal?

Many people choose surgery because they just want their worries about scoliosis to be over. However, scoliosis surgery is not the final solution; merely an irreversible one. Scoliosis can continue to get worse even after spinal fusion, and over 20% of patients require more than one operation.15,16 Furthermore, 40% of patients are legally disabled 16 years after the procedure.17

Long-term evidence suggests that living with a fused spine may be worse than living with a curved one. 38% of patients stated that, if they had the chance to go back in time, they would not have undergone the surgery.18 76% of patients suffer from back pain after 10 years.16 After 15 years, patients report increased difficulty sitting, standing, carrying, bending at the waist, participating in sports, lying on their backs or sides, lifting, performing household chores, and driving a car.20 In every patient who undergoes spinal fusion surgery, there is a permanent loss of spinal flexibility & function.21

The documented risks of scoliosis surgery are bone fragments or instrumentation penetrating into the spinal canal; breakage of the implants; and, compression of the spinal nerves.22 This can lead to neurological deficits such as partial or total paraplegia, quadriplegia, or peripheral nerve damage - which may occur immediately after the operation, or as much as 10 years later.23

Surgery does not reduce rib deformity; instead, thoracoplasty (shaving down the ribs) or rib removal is often recommended for this purpose. This can result in a serious & permanent impairment of normal lung function, and can in fact cause the scoliotic curvature to progress.24 Even if the rib hump does improve after spinal fusion, in the majority of patients, the improvement is temporary, and eventually the situation is worse than it was before.25

The truth is, spinal surgery is an invasive and dangerous procedure, and one that should only be undertaken after all other options have been exhausted. Unfortunately, it is increasingly being recommended as the first resort for children with progressive scoliosis and adults with painful scoliosis. Once done, it cannot be undone; to operate or not is an important decision, and all factors should be considered carefully before committing to spinal fusion surgery.26

References:

15) Danielsson, AJ, Nachemson, AL. Radiologic findings and curve progression 22 years after treatment for adolescent idiopathic scoliosis: comparison of brace and surgical treatment with matching control group of straight individuals. Spine 2001; 26:516.
16) Connolly PJ, Von Schroeder HP, Johnson GE, Kostuik JP. Adolescent idiopathic scoliosis (AIS): long-term effect of instrumentation extending to the lumbar spine. Journal of Bone and Joint Surgery 1995;77-A:1210-1216.
17) Gotze C et al: Long-term results of quality of life in patients with idiopathic scoliosis after Harrington instrumentantion and their relevance for expert evidence. Z Orthop Ihre Grenzgeb 2002;140(5):492-498.
18) Lenke et al: Radiographic results of arthrodesis with Cotrel-Dubousset instrumentation for the treatment of adolescent idiopathic scoliosis: a five to ten year follow-up. J Bone Joint Surg Am 1998;80(6):807-814.
19) Dickson JH, Erwin WD, Rossi D. Harrington instrumentation and arthrodesis for idiopathic scoliosis; a 21-year follow-up. J Bone Joint Surg 1990;72-A:678-690.
20) Dickson JH, Erwin WD, Rossi D. Harrington instrumentation and arthrodesis for idiopathic scoliosis; a 21-year follow-up. J Bone Joint Surg 1990;72-A:678-690.
21) Moreland MS. Outcomes of scoliosis fusion - is stiff and straight better? Stud Health Tech Inform 2002;91:492-497.
22) Hawes M: Impact of spine surgery on signs & symptoms of spinal deformity. Ped Rehab 2006;9(4):318-339.
23) Rittmeister M et al: Cauda equina compression due to a laminar hook: a late complication of posterior instrumentation in scoliosis surgery. Eur Spine J 1999;8:208-210.
24) Hall JE: Spinal surgery before and after Paul Harrington. Spine 1998;23:1356-1361.
25) Weiss HR, Goodall D: Rate of complications in scoliosis surgery - a systematic review of the Pub Med literature. Scoliosis 2008;3:9.
26) Weiss et al: Adolescent idiopathic scoliosis - To operate or not? A debate article. Patient Safety in Surgery 2008;2:25.
Many people choose surgery because they just want their worries about scoliosis to be over. However, scoliosis surgery is not the final solution; merely an irreversible one. Scoliosis can continue to get worse even after spinal fusion, and over 20% of patients require more than one operation.15,16 Furthermore, 40% of patients are legally disabled 16 years after the procedure.17

Long-term evidence suggests that living with a fused spine may be worse than living with a curved one. 38% of patients stated that, if they had the chance to go back in time, they would not have undergone the surgery.18 76% of patients suffer from back pain after 10 years.16 After 15 years, patients report increased difficulty sitting, standing, carrying, bending at the waist, participating in sports, lying on their backs or sides, lifting, performing household chores, and driving a car.20 In every patient who undergoes spinal fusion surgery, there is a permanent loss of spinal flexibility & function.21

The documented risks of scoliosis surgery are bone fragments or instrumentation penetrating into the spinal canal; breakage of the implants; and, compression of the spinal nerves.22 This can lead to neurological deficits such as partial or total paraplegia, quadriplegia, or peripheral nerve damage - which may occur immediately after the operation, or as much as 10 years later.23

Surgery does not reduce rib deformity; instead, thoracoplasty (shaving down the ribs) or rib removal is often recommended for this purpose. This can result in a serious & permanent impairment of normal lung function, and can in fact cause the scoliotic curvature to progress.24 Even if the rib hump does improve after spinal fusion, in the majority of patients, the improvement is temporary, and eventually the situation is worse than it was before.25

The truth is, spinal surgery is an invasive and dangerous procedure, and one that should only be undertaken after all other options have been exhausted. Unfortunately, it is increasingly being recommended as the first resort for children with progressive scoliosis and adults with painful scoliosis. Once done, it cannot be undone; to operate or not is an important decision, and all factors should be considered carefully before committing to spinal fusion surgery.26

References:

15) Danielsson, AJ, Nachemson, AL. Radiologic findings and curve progression 22 years after treatment for adolescent idiopathic scoliosis: comparison of brace and surgical treatment with matching control group of straight individuals. Spine 2001; 26:516.
16) Connolly PJ, Von Schroeder HP, Johnson GE, Kostuik JP. Adolescent idiopathic scoliosis (AIS): long-term effect of instrumentation extending to the lumbar spine. Journal of Bone and Joint Surgery 1995;77-A:1210-1216.
17) Gotze C et al: Long-term results of quality of life in patients with idiopathic scoliosis after Harrington instrumentantion and their relevance for expert evidence. Z Orthop Ihre Grenzgeb 2002;140(5):492-498.
18) Lenke et al: Radiographic results of arthrodesis with Cotrel-Dubousset instrumentation for the treatment of adolescent idiopathic scoliosis: a five to ten year follow-up. J Bone Joint Surg Am 1998;80(6):807-814.
19) Dickson JH, Erwin WD, Rossi D. Harrington instrumentation and arthrodesis for idiopathic scoliosis; a 21-year follow-up. J Bone Joint Surg 1990;72-A:678-690.
20) Dickson JH, Erwin WD, Rossi D. Harrington instrumentation and arthrodesis for idiopathic scoliosis; a 21-year follow-up. J Bone Joint Surg 1990;72-A:678-690.
21) Moreland MS. Outcomes of scoliosis fusion - is stiff and straight better? Stud Health Tech Inform 2002;91:492-497.
22) Hawes M: Impact of spine surgery on signs & symptoms of spinal deformity. Ped Rehab 2006;9(4):318-339.
23) Rittmeister M et al: Cauda equina compression due to a laminar hook: a late complication of posterior instrumentation in scoliosis surgery. Eur Spine J 1999;8:208-210.
24) Hall JE: Spinal surgery before and after Paul Harrington. Spine 1998;23:1356-1361.
25) Weiss HR, Goodall D: Rate of complications in scoliosis surgery - a systematic review of the Pub Med literature. Scoliosis 2008;3:9.
26) Weiss et al: Adolescent idiopathic scoliosis - To operate or not? A debate article. Patient Safety in Surgery 2008;2:25.
Read More


Scoliosis Surgery - Medically Necessary or High Invasive Cosmetic Procedure?

The common approach and reasoning behind prescribing scoliosis surgery is questionable at best. The current system relies on a single measurement called a Cobb angle in order to justify a highly invasive, life altering scoliosis surgery that cost upwards of $250,000-$350,000. In a time where even the most basic of health care services are on the verge of being rationed.

The reasoning and justification for scoliosis surgery is often based on the "assumption" that if scoliosis surgery isn't performed the patient could have (not necessarily will have) problems with their pulmonary system (ability to breathe). This is a flawed logic, because it is known that life-threatening pulmonary scoliosis is extremely rare (in the absence of co-morbitities) if the severe curvature developed after the age of 5 years old (severe scoliosis could cause a heart/lung condition called Cor Pulmonale if the lung volume is compromised prior to full development which is complete by the age 5 in virtually all children). In fact, every single study on scoliosis surgery clearly shows the procedure does NOT improve lung function and can even significantly decrease lung function for up to 2 years post scoliosis surgery.

Chronic pain is another common justification for scoliosis surgery does not eliminate pain. Pain is not an indication for scoliosis surgery and many studies find that many patients are actually in more pain 3-5 years post op than pre-op. Don't believe me? Check out what I dub "the most unhappy place on earth" which is the scoliosis surgery revision section of the National Scoliosis Foundation Scoliosis Forum.

Many adult scoliosis patients exhibit signs and symptoms of decreasing quality of life measures, but all of the research shows scoliosis surgery does not improve quality of life. In fact, when asked if scoliosis surgery benefits the patient researcher Berven stated in the September 2007 SPINE Journal "there are no current, definitive studies that answer the question posed above." Which is odd, because a 17 year post scoliosis surgery follow-up study found 40% of the post scoliosis surgery patients were legally defined as "severely handicapped". This is in sharp contrast with the 50 year follow up study of un-treated scoliosis patients who seemed to have an significantly increased quality of life than many of the post scoliosis surgery patients.

All to often scoliosis surgery is considered as a last ditch effort to halt curve progression, but a closer examination of the data shows scoliosis surgery does not necessarily halt curve progression in adulthood. The average curve progression rate in adults with un-treated idiopathic scoliosis is 1-3 degrees a year. Post scoliosis surgery studies indicate a rate of curve progression in post scoliosis surgery adults at Initial average loss of correction post scoliosis surgery is 3.2 degrees the first year, 6.5 degrees after two years, and 1.0 degrees every year after that of the course of the patient's life.

I believe Dr. Paul Harrington, known for inventing the scoliosis surgery that implants metal rods in scoliotic spines, stated in 1963, "metal does not cure the disease of scoliosis, which is a condition involving much more than the spinal column".

I highly encourage every scoliosis patient whom is considering any scoliosis spine treatment to dig deep into the research available (both pro and con), ask your doctor as many questions as you can think of, and have your x-rays read by at least 2 radiologist (non-surgeons), because they are unbias and the cobb angle measurement (used to determine the "need" for scoliosis surgery for some reason) has a inter-examiner measurement error of /-5-10 degrees.

Everyone has the right to make a truly informed decision.
The common approach and reasoning behind prescribing scoliosis surgery is questionable at best. The current system relies on a single measurement called a Cobb angle in order to justify a highly invasive, life altering scoliosis surgery that cost upwards of $250,000-$350,000. In a time where even the most basic of health care services are on the verge of being rationed.

The reasoning and justification for scoliosis surgery is often based on the "assumption" that if scoliosis surgery isn't performed the patient could have (not necessarily will have) problems with their pulmonary system (ability to breathe). This is a flawed logic, because it is known that life-threatening pulmonary scoliosis is extremely rare (in the absence of co-morbitities) if the severe curvature developed after the age of 5 years old (severe scoliosis could cause a heart/lung condition called Cor Pulmonale if the lung volume is compromised prior to full development which is complete by the age 5 in virtually all children). In fact, every single study on scoliosis surgery clearly shows the procedure does NOT improve lung function and can even significantly decrease lung function for up to 2 years post scoliosis surgery.

Chronic pain is another common justification for scoliosis surgery does not eliminate pain. Pain is not an indication for scoliosis surgery and many studies find that many patients are actually in more pain 3-5 years post op than pre-op. Don't believe me? Check out what I dub "the most unhappy place on earth" which is the scoliosis surgery revision section of the National Scoliosis Foundation Scoliosis Forum.

Many adult scoliosis patients exhibit signs and symptoms of decreasing quality of life measures, but all of the research shows scoliosis surgery does not improve quality of life. In fact, when asked if scoliosis surgery benefits the patient researcher Berven stated in the September 2007 SPINE Journal "there are no current, definitive studies that answer the question posed above." Which is odd, because a 17 year post scoliosis surgery follow-up study found 40% of the post scoliosis surgery patients were legally defined as "severely handicapped". This is in sharp contrast with the 50 year follow up study of un-treated scoliosis patients who seemed to have an significantly increased quality of life than many of the post scoliosis surgery patients.

All to often scoliosis surgery is considered as a last ditch effort to halt curve progression, but a closer examination of the data shows scoliosis surgery does not necessarily halt curve progression in adulthood. The average curve progression rate in adults with un-treated idiopathic scoliosis is 1-3 degrees a year. Post scoliosis surgery studies indicate a rate of curve progression in post scoliosis surgery adults at Initial average loss of correction post scoliosis surgery is 3.2 degrees the first year, 6.5 degrees after two years, and 1.0 degrees every year after that of the course of the patient's life.

I believe Dr. Paul Harrington, known for inventing the scoliosis surgery that implants metal rods in scoliotic spines, stated in 1963, "metal does not cure the disease of scoliosis, which is a condition involving much more than the spinal column".

I highly encourage every scoliosis patient whom is considering any scoliosis spine treatment to dig deep into the research available (both pro and con), ask your doctor as many questions as you can think of, and have your x-rays read by at least 2 radiologist (non-surgeons), because they are unbias and the cobb angle measurement (used to determine the "need" for scoliosis surgery for some reason) has a inter-examiner measurement error of /-5-10 degrees.

Everyone has the right to make a truly informed decision.
Read More


Phys Ed: More Bone (and Less Fat) Through Exercise

For those requiring additional reasons to show up at the running path or at the gym in the dreary heart of winter, science has come up with a compelling new motivation. Exercise can, it appears, keep your bone marrow from becoming too flabby.
This idea is the focus of a series of intriguing recent experiments by Janet Rubin, a professor of medicine at the University of North Carolina and other researchers. For the work, scientists removed bone marrow cells from mice and cultured them. The cells in question, mesenchymal stem cells, are found in bone marrow in both animals and people, waiting for certain molecular signals to tell them to transform into either bone cells, fat cells or, less commonly, something else.
After a stem cell differentiates, of course, it can no longer be anything else: once a fat cell, always a fat cell; once a bone cell, etc. So the fate of marrow stem cells determines the strength and quality of the bone. If a stem cell becomes a fat cell, then the portion of the skeleton to which it might have migrated as a bone cell will be that littlest bit punier. In a study published late last year by researchers at the University of Southern California, the femurs of healthy adults, some in their 20s, others past age 55, were scanned with magnetic resonance imaging. The researchers found that, in both young and old, the amount of fat in the leg’s bone marrow was inversely related to the amount of bone. The more fat in the marrow, the less bone in the thigh.
But what drives a particular stem cell to become a fat cell instead of a bone cell, and does exercise play a role? Earlier experiments by Clinton Rubin, Janet Rubin’s brother and the director of the Center for Biotechnology at Stony Brook University, had shown that mice placed on platforms that were gently vibrating — in an approximation of the forces generated by the muscular contractions of a gentle stroll — developed more bone density than mice who just sat around. Closer examination of the marrow in these experimental mice found that specific genes and gene transcription factors had been stimulated by the vibrations and had, in turn, directed the stem cells to transform into bone.
Something similar happened when Janet Rubin worked directly with the stem cells themselves, even though she was setting them up to become fat cells. To that end, she and her colleagues bathed them in what she calls “a sweet soup,” a medium infused with extra insulin and other elements that normally would encourage the stem cells to differentiate into fat. “They love to become fat cells,” Dr. Rubin said. “It’s discouragingly easy to nudge them in that direction.”
But when the mesenchymal stem cells were stimulated with mechanical vibrations, when they were, in effect, exercised, they did not all become fat cells. “There was a really striking difference in outcomes,” Dr. Rubin said. Her earlier studies with high-magnitude mechanical signals closely approximated a brisk cellular jog. Now she applied lower-magnitude vibrations twice a day, with a rest period of several hours in between. Once again, the stem cells did not all differentiate into fat, even though their cell medium was highly fat-inducing. Dr. Rubin suspects that complicated issues of biochemical signaling underlay the stem cells’ response to the dual-dose regimen. She is currently completing experiments she says she hopes will clarify the mechanisms involved.
Already, though, the findings would appear to have compelling, real-world implications. If you don’t want fatty bone marrow and unhealthy bones, Dr. Rubin said, consider breaking up moderate-intensity workouts into several sessions interspersed throughout the day. Dr. Rubin herself often now works out twice a day for 30 minutes, rather than, as she once did, for a single hourlong bout. “This is the first time in my career that something I’ve done in the lab has changed how I exercise,” she said.
Many questions remain, of course. It’s not clear, for one, whether fat cells generated in bone marrow remain in the marrow or move around to pad, say, the thighs. It’s also not known how exercise affects stem cells located outside the bone marrow. Can it prevent the birth of fat cells all over the body? In Clinton Rubin’s experiments with mice, the vibrated animals wound up with less overall body fat than the control mice, but the reasons are unknown.
Still, one lesson is indisputable. Don’t sit still more than you need to, Dr. Rubin said, and don’t let your children loll about either. “One of the concerns raised” by these experiments, she said, “is that if you make fat cells when you’re young, then you’ve lost any opportunity to have that particular cell be bone,” and the fat cell will remain just that, for life.
For those requiring additional reasons to show up at the running path or at the gym in the dreary heart of winter, science has come up with a compelling new motivation. Exercise can, it appears, keep your bone marrow from becoming too flabby.
This idea is the focus of a series of intriguing recent experiments by Janet Rubin, a professor of medicine at the University of North Carolina and other researchers. For the work, scientists removed bone marrow cells from mice and cultured them. The cells in question, mesenchymal stem cells, are found in bone marrow in both animals and people, waiting for certain molecular signals to tell them to transform into either bone cells, fat cells or, less commonly, something else.
After a stem cell differentiates, of course, it can no longer be anything else: once a fat cell, always a fat cell; once a bone cell, etc. So the fate of marrow stem cells determines the strength and quality of the bone. If a stem cell becomes a fat cell, then the portion of the skeleton to which it might have migrated as a bone cell will be that littlest bit punier. In a study published late last year by researchers at the University of Southern California, the femurs of healthy adults, some in their 20s, others past age 55, were scanned with magnetic resonance imaging. The researchers found that, in both young and old, the amount of fat in the leg’s bone marrow was inversely related to the amount of bone. The more fat in the marrow, the less bone in the thigh.
But what drives a particular stem cell to become a fat cell instead of a bone cell, and does exercise play a role? Earlier experiments by Clinton Rubin, Janet Rubin’s brother and the director of the Center for Biotechnology at Stony Brook University, had shown that mice placed on platforms that were gently vibrating — in an approximation of the forces generated by the muscular contractions of a gentle stroll — developed more bone density than mice who just sat around. Closer examination of the marrow in these experimental mice found that specific genes and gene transcription factors had been stimulated by the vibrations and had, in turn, directed the stem cells to transform into bone.
Something similar happened when Janet Rubin worked directly with the stem cells themselves, even though she was setting them up to become fat cells. To that end, she and her colleagues bathed them in what she calls “a sweet soup,” a medium infused with extra insulin and other elements that normally would encourage the stem cells to differentiate into fat. “They love to become fat cells,” Dr. Rubin said. “It’s discouragingly easy to nudge them in that direction.”
But when the mesenchymal stem cells were stimulated with mechanical vibrations, when they were, in effect, exercised, they did not all become fat cells. “There was a really striking difference in outcomes,” Dr. Rubin said. Her earlier studies with high-magnitude mechanical signals closely approximated a brisk cellular jog. Now she applied lower-magnitude vibrations twice a day, with a rest period of several hours in between. Once again, the stem cells did not all differentiate into fat, even though their cell medium was highly fat-inducing. Dr. Rubin suspects that complicated issues of biochemical signaling underlay the stem cells’ response to the dual-dose regimen. She is currently completing experiments she says she hopes will clarify the mechanisms involved.
Already, though, the findings would appear to have compelling, real-world implications. If you don’t want fatty bone marrow and unhealthy bones, Dr. Rubin said, consider breaking up moderate-intensity workouts into several sessions interspersed throughout the day. Dr. Rubin herself often now works out twice a day for 30 minutes, rather than, as she once did, for a single hourlong bout. “This is the first time in my career that something I’ve done in the lab has changed how I exercise,” she said.
Many questions remain, of course. It’s not clear, for one, whether fat cells generated in bone marrow remain in the marrow or move around to pad, say, the thighs. It’s also not known how exercise affects stem cells located outside the bone marrow. Can it prevent the birth of fat cells all over the body? In Clinton Rubin’s experiments with mice, the vibrated animals wound up with less overall body fat than the control mice, but the reasons are unknown.
Still, one lesson is indisputable. Don’t sit still more than you need to, Dr. Rubin said, and don’t let your children loll about either. “One of the concerns raised” by these experiments, she said, “is that if you make fat cells when you’re young, then you’ve lost any opportunity to have that particular cell be bone,” and the fat cell will remain just that, for life.
Read More


Release of fully revised and updated version of Your Plan for Natural Scoliosis Prevention and Treatment Book

Second Edition, releasing March 28, 2011, offers promise of straighter spine and improved overall health

The award-winning Dr. Kevin Lau, D.C., announces the release of his new and improved version of Your Plan for Natural Scoliosis Prevention and Treatment: Health In Your Hands on March 27, 2011. The new book expands upon Dr. Lau’s unique school of posture and healing introduced in his comprehensive first edition.
“Modern medicine, alone, has failed to effectively manage the condition of Scoliosis,” said Dr. Lau. “The treatment program I designed combines ancient wisdom with modern research and technology, culminating in the best overall results. Today, as a practicing chiropractor and nutritionist, I am more certain than ever that scoliosis, and other disorders, can be completely cured and treated effectively.”

Second Edition, releasing March 28, 2011, offers promise of straighter spine and improved overall health

The award-winning Dr. Kevin Lau, D.C., announces the release of his new and improved version of Your Plan for Natural Scoliosis Prevention and Treatment: Health In Your Hands on March 27, 2011. The new book expands upon Dr. Lau’s unique school of posture and healing introduced in his comprehensive first edition.
“Modern medicine, alone, has failed to effectively manage the condition of Scoliosis,” said Dr. Lau. “The treatment program I designed combines ancient wisdom with modern research and technology, culminating in the best overall results. Today, as a practicing chiropractor and nutritionist, I am more certain than ever that scoliosis, and other disorders, can be completely cured and treated effectively.”

Read More


The Missing Nutrient to Blame for Heart Attacks and Osteoporosis (Nope - NOT Calcium or Vitamin D)

The past decade has brought an explosion of research revealing the many beneficial roles vitamin D plays in keeping you healthy.
But there's a new kid on the block that could end up being "the next vitamin D," and we are finding that some of vitamin D's benefits are greatly enhancedwhen combined with this other vitamin.
That "new kid" is vitamin K.
Much new research is now focusing on the synergy between vitamin K (specifically, vitamin K2) and vitamin D3, particularly in terms of bone strength and cardiovascular health.
Before discussing the influence of each of these vitamins, let's review a bit about vitamins D and K and what their roles are.

Vitamin D: A Brief Review

Vitamin D is a key player in your overall health. The name is misleading -- it isn't actually a vitamin at all but a potent neuroregulatory steroidal hormone that influences nearly 3,000 of your 25,000 genes.
It literally turns on and off genes that can exacerbate -- or prevent -- many diseases. Vitamin D has been shown to influence dozens of conditions, including:
CancerHypertensionHeart disease
AutismObesityRheumatoid arthritis
Diabetes 1 and 2Multiple SclerosisCrohn's disease
Cold & FluInflammatory Bowel DiseaseTuberculosis
SepticemiaSigns of agingDementia
Eczema & PsoriasisInsomniaHearing loss
Muscle painCavitiesPeriodontal disease
OsteoporosisMacular degenerationReduced C-section risk
Pre eclampsiaSeizuresInfertility
AsthmaCystic fibrosisMigraines
DepressionAlzheimer's diseaseSchizophrenia

One of the key factors explaining today's high rates of chronic disease, besides poor diet and sedentary lifestyle, is an epidemic of vitamin D deficiency. It is estimated that 85 percent of Americans have insufficient levels of vitamin D.
Sadly, when the Institute of Medicine's (IOM) Food and Nutrition Board (FNB) released their updated recommendations for vitamin D (and calcium) on November 30, 2010, it caused shockwaves of disappointment through the natural health community.
According to the IOM, the new recommended daily allowance (RDA) for pregnant women and adults up to 70 years of age is the same as that for infants and children -- a measly 600 IU's. This despite the overwhelming evidence showing that vitamin D is extremely important for a wide variety of health conditions besides bone health, and that most people need about ten times this amount or more.
For more information about the IOM's new vitamin D recommendations, please see this recent articlein which Dr. Cannell, founder of the Vitamin D Council, and Carole Baggerly, founder ofGrassrootsHealth, share their concerns.
The best ways to increase your vitamin D levels, in my order of preference, are by:
  1. Exposing your skin natural sunlight. Vitamin D from sunlight acts as a pro-hormone, rapidly converting in your skin into 25-hydroxyvitamin D, or vitamin D3.
  2. Using a safe home tanning bed like the Sun Splash to achieve similar results as that from natural sunlight exposure.
  3. Taking an oral vitamin D3 supplement whenever natural sun exposure is not an option.
For more about vitamin D and how to get the appropriate test of your blood levels, please listen to my free one hour lecture about vitamin D, and refer to my vitamin D resource page.

Vitamin K Basics

Vitamin K may very well end up being as important for you as vitamin D, as research continues to illuminate the growing list of its benefits for your health. Vitamin K is probably where vitamin D was ten years ago, with respect to its appreciation as a vital nutrient that has far more advantages than originally thought.
And, according to Dr. Cees Vermeer, one of the world's top researchers into vitamin K, nearly everyone is deficient in it -- just like most people are deficient in vitamin D.
Most of you get enough K from your diet to maintain adequate blood clotting, but NOT enough to offer protection against the following health problems -- and the list continues to grow:
Arterial calcification, cardiovascular disease and varicose veinsBrain health problems, including dementia (the specifics of which are under study)
OsteoporosisTooth decay
Prostate cancer, lung cancer, liver cancer and leukemiaInfectious diseases such as pneumonia

Vitamin K exists in two basic forms, K1 and K2:
  1. Vitamin K1: Found in green vegetables, K1 goes directly to your liver and helps you maintain a healthy blood clotting system. (This is the kind of K that infants need to help prevent a serious bleeding disorder.)
  2. Vitamin K2: Bacteria produce this type of vitamin K. It is present in high quantities in your gut, but unfortunately is not absorbed from there and passes out in your stool. K2 goes straight to vessel walls, bones, and tissues other than your liver.
Making a long story even longer, there are several different forms of vitamin K2: MK4, MK7, MK8, and MK9. The form of vitamin K that has the most significance for our purposes here is MK7, a newer and longer acting form with more practical applications.
Most vitamin K2 supplements are in the form MK7.
MK7 is extracted from the Japanese fermented soy product called natto. You could actually get loads of MK7 from consuming natto as it is relatively inexpensive, and is available in most Asian food markets. Few people, however, tolerate its smell and slimy texture, so most people who find natto unpalatable prefer to take a supplement. You can also get MK7 by eating fermented cheeses.
For a comprehensive exploration of all the research and functions of vitamin K, refer to this article on the Weston Price website.
Now, how do vitamin D and vitamin K play together?

Vitamins D and K: "The Gatekeeper and the Traffic Cop"

One of the undisputed benefits vitamin D provides for you is improved bone development by helping you ABSORB calcium. This is not news -- we have known about vitamin D and the absorption of calcium for many decades.
But there is new evidence that it is the vitamin K (specifically, vitamin K2) that directs the calcium to your skeleton, while preventing it from being deposited where you don't want it -- i.e., your organs, joint spaces, and arteries. A large part of arterial plaque consists of calcium deposits (atherosclerosis), hence the term "hardening of the arteries."
Vitamin K2 activates a protein hormone called osteocalcin, produced by osteoblasts, which is needed to bind calcium into the matrix of your bone. Osteocalcin also appears to help prevent calcium from depositing into your arteries.
You can think of vitamin D as the gatekeeper, controlling who gets in, and vitamin K as the traffic cop, directing the traffic to where it needs to go.
Lots of traffic -- but no traffic cop -- means clogging, crowding and chaos everywhere!
In other words, without the help of vitamin K2, the calcium that your vitamin D so effectively lets in might be working AGAINST you -- by building up your coronary arteries rather than your bones.
There is even evidence that the safety of vitamin D is dependent on vitamin K, and that vitamin D toxicity (although very rare with the D3 form) is actually caused by vitamin K2 deficiency.

Vitamin K, Vitamin D, and Cardiovascular Disease

When your body's soft tissues are damaged, they respond with an inflammatory process that can result in the deposition of calcium into the damaged tissue. When this occurs in your blood vessels, you have the underlying mechanism of coronary artery disease -- the buildup of plaque -- that can lead you down the path to a heart attack.
Vitamin K and vitamin D work together to increase Matrix GLA Protein (or MGP), the protein responsible for protecting your blood vessels from calcification. In healthy arteries, MGP congregates around the elastic fibers of your tunica media (arterial lining), guarding them against calcium crystal formation.
MGP is so important that it can be used as a laboratory measure of your vascular and cardiac status.
According to Professor Cees Vermeer:
"The only mechanism for arteries to protect themselves from calcification is via the vitamin K-dependent protein MGP. MPG is the most powerful inhibitor of soft tissue calcification presently known, but nonsupplemented healthy adults are insufficient in vitamin K to a level that 30 per cent of their MGP is synthesized in an inactive form.
So, protection against cardiovascular calcification is only 70 per cent in the young, healthy population, and this figure decreases at increasing age."
As you would predict, scientific studies confirm that increased dietary intake of vitamin K2 does indeed reduce your risk for coronary heart disease:
  • In 2004, the Rotterdam study was the first study demonstrating the life-extending effects of vitamin K2. People who had the highest intake of vitamin K2 had 50 percent lower risk of death from coronary heart disease and calcification than people with the lowest intake of vitamin K2.
  • In a subsequent study called the Prospect study, 16,000 people were followed for 10 years. Researchers found that each additional 10 mcg of vitamin K2 in the diet resulted in 9 percent fewer cardiac events.
  • Animal studies show that vitamin K2 not only prevents hardening of the arteries but can actually reverse calcification of highly calcified arteries, by activating MGP.
  • People with severe calcifications have high percentages of inactive osteocalcin, which indicates a general deficiency of vitamin K2.
Let's take a look at how calcium supplements play into all of this.

Does Your Calcium Supplement Actually Increase Your Risk of Heart Attack?

If you take calcium and vitamin D but are deficient in vitamin K, you could be worse off than if you were not taking those supplements at all, as demonstrated by a recent meta-analysis linking calcium supplements to heart attacks.
This study did indeed find that people taking calcium supplements were more prone to heart attacks. However, that doesn't mean that it was the calcium supplements themselves that caused the heart attacks.
Please remember that calcium is only ONE of the players in your bone and heart health.
This meta-analysis looked at studies involving people taking calcium in isolation, without complementary nutrients like magnesium, vitamin D and vitamin K, which help keep your body in balance. In the absence of those other important cofactors, calcium CAN have adverse effects, such as building up in coronary arteries and causing heart attacks, which is really what this analysis detected.
And the FORM of calcium you take matters greatly, which I will address in a moment.
You simply can't take isolated supplements "willy-nilly" and expect to optimize very complicated physiological processes.
Vitamin D itself has been found to protect your heart. A study in the Netherlands provides compelling evidence that a high vitamin D status is associated with improved survival in heart failure patients.
If you are going to take calcium, you need to balance it out with vitamin D and vitamin K, at the very least. It is also important that you get adequate magnesium, silica, omega-3 fatty acids, and weight-bearing exercise, which are all important to the health of your bones.
Which leads us to the next important topic: osteoporosis.

Dense Bones are NOT Necessarily Strong Bones

One of the great health concerns for menopausal women is osteoporosis.
The classic way osteopenia (decreased bone density) and osteoporosis are diagnosed is by an x-ray called a DEXA scan, which specifically measures bone density, or the degree of mineralization of your bones.
But bone strength is MORE than bone density -- which is why drugs such as biphosphinates have failed so miserably.
Your bones are made up of minerals in a collagen matrix. The minerals give your bones rigidity and density, but the collagen gives your bones flexibility. Without good flexibility, they become brittle and break easily.
So, density does NOT equal strength!
Drugs like Fosamax build up a lot of minerals and make the bone LOOK very dense, but in reality, they are extremely brittle and prone to fracture, which is why there have been so many cases of hip fracture among people taking these damaging drugs.
Biphosphinates are poisons that destroy your osteoclasts, which interferes with your normal bone-remodeling process.
You are much better off building your bones using exercise and nutritional therapies, hormones like progesterone and vitamins D and K.

The Calcium Myth: Revising Our Theory of Bone Mineralization

Countries with the highest calcium consumption have the highest rates of osteoporosis -- namely, the U.S., Canada and Scandinavian countries. This is commonly known as the "calcium paradox."
This is because nutritional guidelines have been based on an incorrect theory of bone mineralization.
When you take the wrong form of calcium, or when your body's ability to direct calcium to the right places becomes impaired (as when you are deficient in vitamin K), calcium is deposited where it shouldn't be -- like sand in gears.
David Wolfe, raw food "guru" and nutritionist, has extensively studied and written about calcium physiology. In an interview, Wolfe discusses the myth about calcium's role in bone health:
"Calcium does not build bones, and that is one of the biggest misconceptions ever, and it actually goes to the real core of our problems with science. That is, the human body is a complex biological machine and an unbelievable mystery.
There is strong evidence that if you eat calcium—let's say it's calcium from coral calcium, for example, oyster shell calcium—it is almost impossible to get that into your bones to increase bone density. The amount of increased bone density, at best, is 1 or 2 percent. That is not good enough."
He goes on to explain that many calcium supplements (such as coral calcium, oyster shell calcium, calcium citrate, and calcium carbonate) are not well handled by your body. These forms of calcium can't be broken down and form miniature "rocks" that get deposited in your soft tissues -- kind of like hard water calcium deposits in your showerhead that eventually impede the flow.
According to Wolfe and others, these silty calcium deposits are major contributors and even causative factors in many conditions, including:
AgingGallstones, colon cancer and Crohn's disease
Coronary artery disease and atherosclerosisKidney stones
Dental plaque and gum diseaseOvarian cysts
HypothyroidismCataracts, glaucoma, and macular degeneration
Obesity and diabetesBone spurs, stiff joints, osteoarthritis, tendonitis and bone cancer
Alzheimer's diseaseCellulite and scar tissue
Breast cancer and cysts (fibrocystic breasts) 

How Vitamin K Prevents You from Turning into a Walking "Coral Reef"

Complicating the problem of calcium deposits are nanobacteria that actually use this bad calcium to their advantage, forming hard shells of calcium phosphate that serve as defensive armor against your body's immune system, like a clamshell shields a clam.
When the shells harden, toxins such as mercury, pesticides, and plastics are trapped in there, which is why it is so hard for you to get those toxins out of your body. This encapsulated space also forms an excellent hiding area for opportunistic viruses, bacteria and fungi.
Overconsumption of calcium creates other mineral deficiencies and imbalances, and Western civilization vastly overconsumes calcium.
According to Rudolf Steiner, creator of biodynamic farming:
"Most calcium is gravitational and channels or conducts forces which accelerate gravity. The more calcium supplements one takes, the quicker one is dragged back into the Earth for recycling."
You don't want to turn into a human coral reef -- or be "recycled into the Earth" before your time!
By getting adequate vitamins D and K, your body will work synergistically to escort calcium to where it's needed, while preventing it from accumulating where it shouldn't.

So, What REALLY Increases Bone Strength?

As it turns out, you need a combination of plant-derived minerals for strong bones.
Your bones are actually composed of at least a dozen minerals. If you just focus on calcium, you will likely weaken your bones and increase your risk of osteoporosis as Dr. Robert Thompson explains in his book, The Calcium Lie.
It's more likely your body can use calcium correctly if it's plant-derived calcium. Good sources include raw milk from pasture-raised cows (who eat the plants), leafy green vegetables, the pith of citrus fruits, carob, and wheatgrass, to name a few.
But you also need sources of silica and magnesium, which some researchers say is actually enzymatically "transmuted" by your body into the kind of calcium your bones can use. This theory was first put forth by French scientist Louis Kevran, a Nobel Prize nominee who spent years studying how silica and calcium are related.
Good sources of silica are cucumbers, bell peppers, tomatoes, and a number of herbs including horsetail, nettles, oat straw, and alfalfa.
The absolute best source of magnesium is raw organic cacao. Yes, healthy high quality chocolate is extremely rich in magnesium!
A great source of trace minerals, which are important for many of your body's functions, is Himalayan Crystal Salt, which contains all 84 elements found in your body.
The bottom line?
The absolute best way to achieve healthy bones is a diet rich in fresh, raw whole foods that maximizes natural minerals so that your body has the raw materials it needs to do what it was designed to do.
Of course, the same can be said for your heart -- or any other part of your body.

To Wrap it Up ...

In order to achieve the best possible health, you have to devise a plan of attack from multiple angles. These suggestions will synergistically help keep your heart, blood vessels, organs and bones healthy.
  1. Optimize your vitamin D either from natural sunlight exposure or an oral supplement, as described above. Check your blood levels regularly.
  2. Optimize your vitamin K through a combination of dietary sources (leafy green vegetables, fermented foods like natto, raw milk cheeses, etc.) and a K2 supplement, if needed. Although the exact dosing (for oral supplementation) is yet to be determined, Dr. Vermeer recommends up to 185 mcg daily for adults. You must use caution on the higher doses if you take anticoagulants, but if you are generally healthy and not on these types of medications, I suggest 150-300 mcg daily.
  3. Make sure you do weight-bearing exercise, which has profound benefits to both your skeletal and cardiovascular systems. My favorite is the Peak Fitness system but it is also very important to do strength training exercises to produce the dynamic electric forces in your bones which will stimulate the osteoblasts in your bone to produce new bone..
  4. Consume a wide variety of fresh, local organic whole foods, including vegetables, fruits, nuts, seeds, organic meats and eggs, and raw organic unpasteurized dairy. The more of your diet you consume RAW, the better nourished you will be. Minimize sugar and refined grains.
  5. Consider a high-quality animal source of omega-3 fatty acid supplement—my favorite is krill oil.
  6. Make sure you are getting enough restorative sleep each night.
  7. Handle the stress in your life since it has a significant impact on your physical and mental well-being. My favorite de-stressing tool is the Emotional Freedom Technique (EFT).
The past decade has brought an explosion of research revealing the many beneficial roles vitamin D plays in keeping you healthy.
But there's a new kid on the block that could end up being "the next vitamin D," and we are finding that some of vitamin D's benefits are greatly enhancedwhen combined with this other vitamin.
That "new kid" is vitamin K.
Much new research is now focusing on the synergy between vitamin K (specifically, vitamin K2) and vitamin D3, particularly in terms of bone strength and cardiovascular health.
Before discussing the influence of each of these vitamins, let's review a bit about vitamins D and K and what their roles are.

Vitamin D: A Brief Review

Vitamin D is a key player in your overall health. The name is misleading -- it isn't actually a vitamin at all but a potent neuroregulatory steroidal hormone that influences nearly 3,000 of your 25,000 genes.
It literally turns on and off genes that can exacerbate -- or prevent -- many diseases. Vitamin D has been shown to influence dozens of conditions, including:
CancerHypertensionHeart disease
AutismObesityRheumatoid arthritis
Diabetes 1 and 2Multiple SclerosisCrohn's disease
Cold & FluInflammatory Bowel DiseaseTuberculosis
SepticemiaSigns of agingDementia
Eczema & PsoriasisInsomniaHearing loss
Muscle painCavitiesPeriodontal disease
OsteoporosisMacular degenerationReduced C-section risk
Pre eclampsiaSeizuresInfertility
AsthmaCystic fibrosisMigraines
DepressionAlzheimer's diseaseSchizophrenia

One of the key factors explaining today's high rates of chronic disease, besides poor diet and sedentary lifestyle, is an epidemic of vitamin D deficiency. It is estimated that 85 percent of Americans have insufficient levels of vitamin D.
Sadly, when the Institute of Medicine's (IOM) Food and Nutrition Board (FNB) released their updated recommendations for vitamin D (and calcium) on November 30, 2010, it caused shockwaves of disappointment through the natural health community.
According to the IOM, the new recommended daily allowance (RDA) for pregnant women and adults up to 70 years of age is the same as that for infants and children -- a measly 600 IU's. This despite the overwhelming evidence showing that vitamin D is extremely important for a wide variety of health conditions besides bone health, and that most people need about ten times this amount or more.
For more information about the IOM's new vitamin D recommendations, please see this recent articlein which Dr. Cannell, founder of the Vitamin D Council, and Carole Baggerly, founder ofGrassrootsHealth, share their concerns.
The best ways to increase your vitamin D levels, in my order of preference, are by:
  1. Exposing your skin natural sunlight. Vitamin D from sunlight acts as a pro-hormone, rapidly converting in your skin into 25-hydroxyvitamin D, or vitamin D3.
  2. Using a safe home tanning bed like the Sun Splash to achieve similar results as that from natural sunlight exposure.
  3. Taking an oral vitamin D3 supplement whenever natural sun exposure is not an option.
For more about vitamin D and how to get the appropriate test of your blood levels, please listen to my free one hour lecture about vitamin D, and refer to my vitamin D resource page.

Vitamin K Basics

Vitamin K may very well end up being as important for you as vitamin D, as research continues to illuminate the growing list of its benefits for your health. Vitamin K is probably where vitamin D was ten years ago, with respect to its appreciation as a vital nutrient that has far more advantages than originally thought.
And, according to Dr. Cees Vermeer, one of the world's top researchers into vitamin K, nearly everyone is deficient in it -- just like most people are deficient in vitamin D.
Most of you get enough K from your diet to maintain adequate blood clotting, but NOT enough to offer protection against the following health problems -- and the list continues to grow:
Arterial calcification, cardiovascular disease and varicose veinsBrain health problems, including dementia (the specifics of which are under study)
OsteoporosisTooth decay
Prostate cancer, lung cancer, liver cancer and leukemiaInfectious diseases such as pneumonia

Vitamin K exists in two basic forms, K1 and K2:
  1. Vitamin K1: Found in green vegetables, K1 goes directly to your liver and helps you maintain a healthy blood clotting system. (This is the kind of K that infants need to help prevent a serious bleeding disorder.)
  2. Vitamin K2: Bacteria produce this type of vitamin K. It is present in high quantities in your gut, but unfortunately is not absorbed from there and passes out in your stool. K2 goes straight to vessel walls, bones, and tissues other than your liver.
Making a long story even longer, there are several different forms of vitamin K2: MK4, MK7, MK8, and MK9. The form of vitamin K that has the most significance for our purposes here is MK7, a newer and longer acting form with more practical applications.
Most vitamin K2 supplements are in the form MK7.
MK7 is extracted from the Japanese fermented soy product called natto. You could actually get loads of MK7 from consuming natto as it is relatively inexpensive, and is available in most Asian food markets. Few people, however, tolerate its smell and slimy texture, so most people who find natto unpalatable prefer to take a supplement. You can also get MK7 by eating fermented cheeses.
For a comprehensive exploration of all the research and functions of vitamin K, refer to this article on the Weston Price website.
Now, how do vitamin D and vitamin K play together?

Vitamins D and K: "The Gatekeeper and the Traffic Cop"

One of the undisputed benefits vitamin D provides for you is improved bone development by helping you ABSORB calcium. This is not news -- we have known about vitamin D and the absorption of calcium for many decades.
But there is new evidence that it is the vitamin K (specifically, vitamin K2) that directs the calcium to your skeleton, while preventing it from being deposited where you don't want it -- i.e., your organs, joint spaces, and arteries. A large part of arterial plaque consists of calcium deposits (atherosclerosis), hence the term "hardening of the arteries."
Vitamin K2 activates a protein hormone called osteocalcin, produced by osteoblasts, which is needed to bind calcium into the matrix of your bone. Osteocalcin also appears to help prevent calcium from depositing into your arteries.
You can think of vitamin D as the gatekeeper, controlling who gets in, and vitamin K as the traffic cop, directing the traffic to where it needs to go.
Lots of traffic -- but no traffic cop -- means clogging, crowding and chaos everywhere!
In other words, without the help of vitamin K2, the calcium that your vitamin D so effectively lets in might be working AGAINST you -- by building up your coronary arteries rather than your bones.
There is even evidence that the safety of vitamin D is dependent on vitamin K, and that vitamin D toxicity (although very rare with the D3 form) is actually caused by vitamin K2 deficiency.

Vitamin K, Vitamin D, and Cardiovascular Disease

When your body's soft tissues are damaged, they respond with an inflammatory process that can result in the deposition of calcium into the damaged tissue. When this occurs in your blood vessels, you have the underlying mechanism of coronary artery disease -- the buildup of plaque -- that can lead you down the path to a heart attack.
Vitamin K and vitamin D work together to increase Matrix GLA Protein (or MGP), the protein responsible for protecting your blood vessels from calcification. In healthy arteries, MGP congregates around the elastic fibers of your tunica media (arterial lining), guarding them against calcium crystal formation.
MGP is so important that it can be used as a laboratory measure of your vascular and cardiac status.
According to Professor Cees Vermeer:
"The only mechanism for arteries to protect themselves from calcification is via the vitamin K-dependent protein MGP. MPG is the most powerful inhibitor of soft tissue calcification presently known, but nonsupplemented healthy adults are insufficient in vitamin K to a level that 30 per cent of their MGP is synthesized in an inactive form.
So, protection against cardiovascular calcification is only 70 per cent in the young, healthy population, and this figure decreases at increasing age."
As you would predict, scientific studies confirm that increased dietary intake of vitamin K2 does indeed reduce your risk for coronary heart disease:
  • In 2004, the Rotterdam study was the first study demonstrating the life-extending effects of vitamin K2. People who had the highest intake of vitamin K2 had 50 percent lower risk of death from coronary heart disease and calcification than people with the lowest intake of vitamin K2.
  • In a subsequent study called the Prospect study, 16,000 people were followed for 10 years. Researchers found that each additional 10 mcg of vitamin K2 in the diet resulted in 9 percent fewer cardiac events.
  • Animal studies show that vitamin K2 not only prevents hardening of the arteries but can actually reverse calcification of highly calcified arteries, by activating MGP.
  • People with severe calcifications have high percentages of inactive osteocalcin, which indicates a general deficiency of vitamin K2.
Let's take a look at how calcium supplements play into all of this.

Does Your Calcium Supplement Actually Increase Your Risk of Heart Attack?

If you take calcium and vitamin D but are deficient in vitamin K, you could be worse off than if you were not taking those supplements at all, as demonstrated by a recent meta-analysis linking calcium supplements to heart attacks.
This study did indeed find that people taking calcium supplements were more prone to heart attacks. However, that doesn't mean that it was the calcium supplements themselves that caused the heart attacks.
Please remember that calcium is only ONE of the players in your bone and heart health.
This meta-analysis looked at studies involving people taking calcium in isolation, without complementary nutrients like magnesium, vitamin D and vitamin K, which help keep your body in balance. In the absence of those other important cofactors, calcium CAN have adverse effects, such as building up in coronary arteries and causing heart attacks, which is really what this analysis detected.
And the FORM of calcium you take matters greatly, which I will address in a moment.
You simply can't take isolated supplements "willy-nilly" and expect to optimize very complicated physiological processes.
Vitamin D itself has been found to protect your heart. A study in the Netherlands provides compelling evidence that a high vitamin D status is associated with improved survival in heart failure patients.
If you are going to take calcium, you need to balance it out with vitamin D and vitamin K, at the very least. It is also important that you get adequate magnesium, silica, omega-3 fatty acids, and weight-bearing exercise, which are all important to the health of your bones.
Which leads us to the next important topic: osteoporosis.

Dense Bones are NOT Necessarily Strong Bones

One of the great health concerns for menopausal women is osteoporosis.
The classic way osteopenia (decreased bone density) and osteoporosis are diagnosed is by an x-ray called a DEXA scan, which specifically measures bone density, or the degree of mineralization of your bones.
But bone strength is MORE than bone density -- which is why drugs such as biphosphinates have failed so miserably.
Your bones are made up of minerals in a collagen matrix. The minerals give your bones rigidity and density, but the collagen gives your bones flexibility. Without good flexibility, they become brittle and break easily.
So, density does NOT equal strength!
Drugs like Fosamax build up a lot of minerals and make the bone LOOK very dense, but in reality, they are extremely brittle and prone to fracture, which is why there have been so many cases of hip fracture among people taking these damaging drugs.
Biphosphinates are poisons that destroy your osteoclasts, which interferes with your normal bone-remodeling process.
You are much better off building your bones using exercise and nutritional therapies, hormones like progesterone and vitamins D and K.

The Calcium Myth: Revising Our Theory of Bone Mineralization

Countries with the highest calcium consumption have the highest rates of osteoporosis -- namely, the U.S., Canada and Scandinavian countries. This is commonly known as the "calcium paradox."
This is because nutritional guidelines have been based on an incorrect theory of bone mineralization.
When you take the wrong form of calcium, or when your body's ability to direct calcium to the right places becomes impaired (as when you are deficient in vitamin K), calcium is deposited where it shouldn't be -- like sand in gears.
David Wolfe, raw food "guru" and nutritionist, has extensively studied and written about calcium physiology. In an interview, Wolfe discusses the myth about calcium's role in bone health:
"Calcium does not build bones, and that is one of the biggest misconceptions ever, and it actually goes to the real core of our problems with science. That is, the human body is a complex biological machine and an unbelievable mystery.
There is strong evidence that if you eat calcium—let's say it's calcium from coral calcium, for example, oyster shell calcium—it is almost impossible to get that into your bones to increase bone density. The amount of increased bone density, at best, is 1 or 2 percent. That is not good enough."
He goes on to explain that many calcium supplements (such as coral calcium, oyster shell calcium, calcium citrate, and calcium carbonate) are not well handled by your body. These forms of calcium can't be broken down and form miniature "rocks" that get deposited in your soft tissues -- kind of like hard water calcium deposits in your showerhead that eventually impede the flow.
According to Wolfe and others, these silty calcium deposits are major contributors and even causative factors in many conditions, including:
AgingGallstones, colon cancer and Crohn's disease
Coronary artery disease and atherosclerosisKidney stones
Dental plaque and gum diseaseOvarian cysts
HypothyroidismCataracts, glaucoma, and macular degeneration
Obesity and diabetesBone spurs, stiff joints, osteoarthritis, tendonitis and bone cancer
Alzheimer's diseaseCellulite and scar tissue
Breast cancer and cysts (fibrocystic breasts) 

How Vitamin K Prevents You from Turning into a Walking "Coral Reef"

Complicating the problem of calcium deposits are nanobacteria that actually use this bad calcium to their advantage, forming hard shells of calcium phosphate that serve as defensive armor against your body's immune system, like a clamshell shields a clam.
When the shells harden, toxins such as mercury, pesticides, and plastics are trapped in there, which is why it is so hard for you to get those toxins out of your body. This encapsulated space also forms an excellent hiding area for opportunistic viruses, bacteria and fungi.
Overconsumption of calcium creates other mineral deficiencies and imbalances, and Western civilization vastly overconsumes calcium.
According to Rudolf Steiner, creator of biodynamic farming:
"Most calcium is gravitational and channels or conducts forces which accelerate gravity. The more calcium supplements one takes, the quicker one is dragged back into the Earth for recycling."
You don't want to turn into a human coral reef -- or be "recycled into the Earth" before your time!
By getting adequate vitamins D and K, your body will work synergistically to escort calcium to where it's needed, while preventing it from accumulating where it shouldn't.

So, What REALLY Increases Bone Strength?

As it turns out, you need a combination of plant-derived minerals for strong bones.
Your bones are actually composed of at least a dozen minerals. If you just focus on calcium, you will likely weaken your bones and increase your risk of osteoporosis as Dr. Robert Thompson explains in his book, The Calcium Lie.
It's more likely your body can use calcium correctly if it's plant-derived calcium. Good sources include raw milk from pasture-raised cows (who eat the plants), leafy green vegetables, the pith of citrus fruits, carob, and wheatgrass, to name a few.
But you also need sources of silica and magnesium, which some researchers say is actually enzymatically "transmuted" by your body into the kind of calcium your bones can use. This theory was first put forth by French scientist Louis Kevran, a Nobel Prize nominee who spent years studying how silica and calcium are related.
Good sources of silica are cucumbers, bell peppers, tomatoes, and a number of herbs including horsetail, nettles, oat straw, and alfalfa.
The absolute best source of magnesium is raw organic cacao. Yes, healthy high quality chocolate is extremely rich in magnesium!
A great source of trace minerals, which are important for many of your body's functions, is Himalayan Crystal Salt, which contains all 84 elements found in your body.
The bottom line?
The absolute best way to achieve healthy bones is a diet rich in fresh, raw whole foods that maximizes natural minerals so that your body has the raw materials it needs to do what it was designed to do.
Of course, the same can be said for your heart -- or any other part of your body.

To Wrap it Up ...

In order to achieve the best possible health, you have to devise a plan of attack from multiple angles. These suggestions will synergistically help keep your heart, blood vessels, organs and bones healthy.
  1. Optimize your vitamin D either from natural sunlight exposure or an oral supplement, as described above. Check your blood levels regularly.
  2. Optimize your vitamin K through a combination of dietary sources (leafy green vegetables, fermented foods like natto, raw milk cheeses, etc.) and a K2 supplement, if needed. Although the exact dosing (for oral supplementation) is yet to be determined, Dr. Vermeer recommends up to 185 mcg daily for adults. You must use caution on the higher doses if you take anticoagulants, but if you are generally healthy and not on these types of medications, I suggest 150-300 mcg daily.
  3. Make sure you do weight-bearing exercise, which has profound benefits to both your skeletal and cardiovascular systems. My favorite is the Peak Fitness system but it is also very important to do strength training exercises to produce the dynamic electric forces in your bones which will stimulate the osteoblasts in your bone to produce new bone..
  4. Consume a wide variety of fresh, local organic whole foods, including vegetables, fruits, nuts, seeds, organic meats and eggs, and raw organic unpasteurized dairy. The more of your diet you consume RAW, the better nourished you will be. Minimize sugar and refined grains.
  5. Consider a high-quality animal source of omega-3 fatty acid supplement—my favorite is krill oil.
  6. Make sure you are getting enough restorative sleep each night.
  7. Handle the stress in your life since it has a significant impact on your physical and mental well-being. My favorite de-stressing tool is the Emotional Freedom Technique (EFT).
Read More


Thursday, March 24, 2011

Lack Of Sleep Linked To Childhood Obesity

Children who do not get adequate sleep may be at an increased risk of becoming obese, according to a new study in the journal Pediatrics.

According to lead researcher Dr. David Gozal of the University of Chicago in Illinois, children who sleep the least could be four times more likely to develop an unhealthy body weight.

"If a child has a tendency to be obese but gets adequate sleep, he is more likely to be protected than if he is not sleeping as much as he needs," Gozal told ABCnews.com. "Catch-up sleep is better than nothing and can help, but we don't think it can offer complete protection."

For the study, researchers used a special sleep monitoring bracelet on 308 children in Louisville, Kentucky. They followed the group, which ranged in ages from 4 to 10 years old, for one week. They found that those who slept the least were 4.2 times more likely to be obese.

"There is growing evidence for a link between sleep duration and childhood obesity. What is new … is that perhaps even more important than sleep duration is the effect of day to day variability of sleep wake timing on weight regulation," added Dr. Phyllis C. Zee, the director of the Sleep Disorders Center at Northwestern Memorial Hospital.
Children who do not get adequate sleep may be at an increased risk of becoming obese, according to a new study in the journal Pediatrics.

According to lead researcher Dr. David Gozal of the University of Chicago in Illinois, children who sleep the least could be four times more likely to develop an unhealthy body weight.

"If a child has a tendency to be obese but gets adequate sleep, he is more likely to be protected than if he is not sleeping as much as he needs," Gozal told ABCnews.com. "Catch-up sleep is better than nothing and can help, but we don't think it can offer complete protection."

For the study, researchers used a special sleep monitoring bracelet on 308 children in Louisville, Kentucky. They followed the group, which ranged in ages from 4 to 10 years old, for one week. They found that those who slept the least were 4.2 times more likely to be obese.

"There is growing evidence for a link between sleep duration and childhood obesity. What is new … is that perhaps even more important than sleep duration is the effect of day to day variability of sleep wake timing on weight regulation," added Dr. Phyllis C. Zee, the director of the Sleep Disorders Center at Northwestern Memorial Hospital.
Read More